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A. Antonelli

Researcher at The Catholic University of America

Publications -  317
Citations -  8365

A. Antonelli is an academic researcher from The Catholic University of America. The author has contributed to research in topics: Medicine & Branching fraction. The author has an hindex of 48, co-authored 254 publications receiving 7459 citations. Previous affiliations of A. Antonelli include Agostino Gemelli University Polyclinic & Catholic University of the Sacred Heart.

Papers
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Journal ArticleDOI

Branching ratios and spectral functions of tau decays: Final ALEPH measurements and physics implications

Stefan Schael, +278 more
- 01 Dec 2005 - 
TL;DR: The full LEP-1 data set collected with the ALEPH detector at the Z pole during 1991-1995 is analyzed in order to measure the tau decay branching fractions as discussed by the authors.
Journal ArticleDOI

The tracking detector of the KLOE experiment

Marco Adinolfi, +110 more
TL;DR: The Drift Chamber for the KLOE experiment at the Frascati φ-factory, DAΦNE, is described in this article, together with a description of the track reconstruction program and of the calibration procedures.
Journal ArticleDOI

The KLOE electromagnetic calorimeter

TL;DR: The KLOE calorimeter as mentioned in this paper is a fine lead-scintillating fiber sampling calorometer with an energy resolution of 5.4% and a time resolution of 56 ps/E (GeV).
Journal ArticleDOI

Search for charged Higgs bosons : combined results using LEP data

Giovanni Abbiendi, +1210 more
TL;DR: In this paper, the results of the four LEP collaborations, ALEPH, DELPHI, L3 and OPAL, were combined within the Two Higgs Doublet Models (2HDMs) for Type I and Type II benchmark scenarios.
Book ChapterDOI

Neurotrophin presence in human coronary atherosclerosis and metabolic syndrome: a role for NGF and BDNF in cardiovascular disease?

TL;DR: The first evidence for an altered presence of NGF, p75NTR and MC in both coronary vascular and subepicardial adipose tissue in human coronary atherosclerosis is provided and a significant decrease in plasma NGF and BDNF levels and an elevated amount of plasma leptin and adipose MC in metabolic syndrome patients is provided.