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Ajit K. Verma

Researcher at University of Wisconsin-Madison

Publications -  146
Citations -  6064

Ajit K. Verma is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Ornithine decarboxylase & Tumor promotion. The author has an hindex of 44, co-authored 131 publications receiving 5846 citations. Previous affiliations of Ajit K. Verma include Amity University.

Papers
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Journal Article

Correlation of the Inhibition by Retinoids of Tumor Promoter-induced Mouse Epidermal Ornithine Decarboxylase Activity and of Skin Tumor Promotion

Ajit K. Verma, +3 more
- 01 Feb 1979 - 
TL;DR: The results indicate that the possible mechanism of prevention of skin papillomas by retinoids involves their ability to inhibit TPA-induced epidermal ornithine decarboxylase activity and the associated elevated putrescine levels.
Journal Article

Inhibition of 7,12-Dimethylbenz(a)anthracene- and N-Nitrosomethylurea-induced Rat Mammary Cancer by Dietary Flavonol Quercetin

Ajit K. Verma, +3 more
- 15 Oct 1988 - 
TL;DR: The effects of dietary supplementation of flavonol quercetin on both 7,12-dimethylbenz( a )anthracene (DMBA)- and N -nitrosomethylurea-induced mammary cancer in female Sprague-Dawley rats were determined as discussed by the authors.
Journal Article

Chemoprevention of spontaneous intestinal adenomas in the Apc Min mouse model by the nonsteroidal anti-inflammatory drug piroxicam

Russell F. Jacoby, +10 more
- 15 Feb 1996 - 
TL;DR: The Min mouse model demonstrates that the nonsteroidal anti-inflammatory drug piroxicam has strong biological and therapeutic effects, potentially useful for prevention of the early adenoma stage of tumor development.
Journal Article

Inhibition by prostaglandin synthesis inhibitors of the induction of epidermal ornithine decarboxylase activity, the accumulation of prostaglandins, and tumor promotion caused by 12-O-tetradecanoylphorbol-13-acetate.

Ajit K. Verma, +2 more
- 01 Feb 1980 - 
TL;DR: The findings that the application of indomethacin prior to TPA treatment inhibits the accumulation of prostaglandins, the induction of ODC activity, and the formation of skin papillomas suggest that TPA-induced O DC activity may be an important component of the mechanism of skin tumor promotion.
Journal ArticleDOI

A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-κB or ODC transactivation

Hsin-Sheng Yang, +6 more
- 08 Feb 2001 - 
TL;DR: Results indicate that Pdcd4 functions as a transformation suppressor, possibly through inhibiting AP-1 activation in combination with other factors such as enhancing NF-κB activation, and may constitute a useful molecular target for cancer prevention.