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Alex A. R. Webb

Researcher at University of Cambridge

Publications -  109
Citations -  9068

Alex A. R. Webb is an academic researcher from University of Cambridge. The author has contributed to research in topics: Circadian clock & Circadian rhythm. The author has an hindex of 43, co-authored 103 publications receiving 7910 citations. Previous affiliations of Alex A. R. Webb include Lancaster University & University of York.

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Plant Circadian Clocks Increase Photosynthesis, Growth, Survival, and Competitive Advantage

TL;DR: It is shown that a substantial photosynthetic advantage is conferred by correct matching of the circadian clock period with that of the external light-dark cycle, which explains why plants gain advantage from circadian control.
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ABI1 Protein Phosphatase 2C Is a Negative Regulator of Abscisic Acid Signaling

TL;DR: Results indicate that a loss of ABI1 PP2C activity leads to an enhanced responsiveness to ABA, and that the wild-type ABA1 phosphatase is a negative regulator of ABA responses.
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The short-period mutant, toc1-1, alters circadian clock regulation of multiple outputs throughout development in Arabidopsis thaliana

TL;DR: A new set of circadian clock-controlled phenotypes for Arabidopsis thaliana are reported, consistent with the likelihood that TOC1 codes for an oscillator component rather than for an element of an input signaling pathway, and indicate that T OC1 acts on or within the clock independently of light input.
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Stimulus-induced oscillations in guard-cell cytosolic-free calcium

TL;DR: A mechanism for encoding the information required to distinguish between a number of different Ca2+-mobilizing stimuli in guard cells, using stimulus-specific patterns of oscillations in [Ca2+]i is suggested.
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Photosynthetic entrainment of the Arabidopsis thaliana circadian clock

TL;DR: It is demonstrated that endogenous oscillations in sugar levels provide metabolic feedback to the circadian oscillator through the morning-expressed gene PSEUDO-RESPONSE REGULATOR 7 (PRR7), and that prr7 mutants are insensitive to the effects of sucrose on the circadian period.