A
Alfredo Berardelli
Researcher at Sapienza University of Rome
Publications - 634
Citations - 33749
Alfredo Berardelli is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Dystonia & Transcranial magnetic stimulation. The author has an hindex of 84, co-authored 583 publications receiving 30313 citations. Previous affiliations of Alfredo Berardelli include University of Cambridge & Harvard University.
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Non-invasive electrical and magnetic stimulation of the brain, spinal cord and roots: basic principles and procedures for routine clinical application. Report of an IFCN committee
Paolo Maria Rossini,A.T. Barker,Alfredo Berardelli,Maria D. Caramia,Giuseppe Caruso,Roger Q. Cracco,Milan R. Dimitrijevic,Mark Hallett,Yoichi Katayama,Carl Hermann Lücking,A. Maertens de Noordhout,C. D. Marsden,N. M. F. Murray,John C. Rothwell,Michael Swash,C. Tomberg +15 more
TL;DR: This year's jurors included A.M.
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Pathophysiology of bradykinesia in parkinson's disease
TL;DR: It is argued that bradykinesia results from a failure of basal ganglia output to reinforce the cortical mechanisms that prepare and execute the commands to move, which leads to particular difficulty with self-paced movements, prolonged reaction times and abnormal pre-movement EEG activity.
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The pathophysiology of primary dystonia.
Alfredo Berardelli,John C. Rothwell,Mark Hallett,Philip D. Thompson,M. Manfredi,C. D. Marsden +5 more
TL;DR: Evidence suggests that primary dystonia results from a functional disturbance of the basal ganglia, particularly in the striatal control of the globus pallidus (and substantia nigra pars reticulata) and abnormal regulation of brainstem and spinal cord inhibitory interneuronal mechanisms.
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Silent period evoked by transcranial stimulation of the human cortex and cervicomedullary junction.
TL;DR: The findings indicate that the first 50 ms of the silent period after TCS are produced mainly by spinal mechanisms such as after‐hyperpolarization and recurrent inhibition of the spinal motoneurones and if descending inhibitory fibres contribute, their contribution is small.
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Sensorimotor integration in movement disorders
TL;DR: Several observations strongly support the idea that sensorimotor integration is impaired in focal dystonia, and underlines the importance of abnormal sensorim motor integration in the pathophysiology of movement disorders.