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Allison J. Doupe
Researcher at University of California, San Francisco
Publications - 67
Citations - 9330
Allison J. Doupe is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Songbird & Vocal learning. The author has an hindex of 42, co-authored 67 publications receiving 8891 citations. Previous affiliations of Allison J. Doupe include University of Southern California.
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BIRDSONG AND HUMAN SPEECH: Common Themes and Mechanisms
TL;DR: Human speech and birdsong have numerous parallels, with striking similarities in how sensory experience is internalized and used to shape vocal outputs, and how learning is enhanced during a critical period of development.
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Spectral-Temporal Receptive Fields of Nonlinear Auditory Neurons Obtained Using Natural Sounds
TL;DR: The results show that the STRF model is an incomplete description of response properties of nonlinear auditory neurons, but that linear receptive fields are still useful models for understanding higher level sensory processing, as long as the STRFs are estimated from the responses to relevant complex stimuli.
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Contributions of an avian basal ganglia–forebrain circuit to real-time modulation of song
TL;DR: A previously unappreciated capacity of the AFP to direct real-time changes in song is demonstrated and frontal cortical and basal ganglia areas may contribute to motor learning by biasing motor output towards desired targets or by introducing stochastic variability required for reinforcement learning.
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What songbirds teach us about learning
TL;DR: With the discovery and investigation of discrete brain structures required for singing, songbirds are providing insights into neural mechanisms of learning and are addressing such basic issues in neuroscience as perceptual and sensorimotor learning, developmental regulation of plasticity, and the control and function of adult neurogenesis.
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Interruption of a basal ganglia-forebrain circuit prevents plasticity of learned vocalizations.
TL;DR: These findings provide evidence that cortical-basal ganglia circuits may participate in the evaluation of sensory feedback during calibration of motor performance, and demonstrate that damage to such circuits can have little effect on previously learned behaviour while conspicuously disrupting the capacity to adaptively modify that behaviour.