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Amulya Nidhi Shrivastava

Researcher at École Normale Supérieure

Publications -  16
Citations -  1037

Amulya Nidhi Shrivastava is an academic researcher from École Normale Supérieure. The author has contributed to research in topics: Receptor & Purinergic receptor. The author has an hindex of 12, co-authored 16 publications receiving 852 citations. Previous affiliations of Amulya Nidhi Shrivastava include Medical University of Vienna & Birla Institute of Technology, Mesra.

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Molecular Biomarkers: Their significance and application in marine pollution monitoring

TL;DR: An overview of the significance of the use of molecular biomarkers as diagnostic and prognostic tools for marine pollution monitoring is presented.
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α‐synuclein assemblies sequester neuronal α3‐Na+/K+‐ATPase and impair Na+ gradient

TL;DR: It is demonstrated that α‐syn assemblies form clusters within the plasma membrane of neurons, resulting in α3‐NKA redistribution and formation of larger nanoclusters and decreasing the efficiency of Na+ extrusion following stimulus.
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Physico-Pathologic Mechanisms Involved in Neurodegeneration: Misfolded Protein-Plasma Membrane Interactions.

TL;DR: Cell-to-cell transfer of misfolded proteins has been proposed for the intra-cerebral propagation of neurodegenerative disorders and opens up new avenues for therapeutics development targeting the initial interactions of deleterious proteins with the plasma membrane or the subsequent pathological signaling.
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Bidirectional Control of Synaptic GABAAR Clustering by Glutamate and Calcium

TL;DR: By quantum dot single-particle tracking, this work characterize a signaling pathway that promotes the stability of GABAA receptor (GABAAR) postsynaptic organization and counteracts the rapid cluster dispersion caused by glutamate-driven NMDA receptor-dependent calcium influx and calcineurin dephosphorylation.
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β-amyloid and ATP-induced diffusional trapping of astrocyte and neuronal metabotropic glutamate type-5 receptors.

TL;DR: An overexpression of mGluR5s in reactive astrocytes surrounding Aβ plaques in brain sections from an Alzheimer's disease mouse model and Aβ oligomers induced an increase in ATP release following activation of astroglial mGlam R5s by its agonist are observed.