A
Anders Björklund
Researcher at Lund University
Publications - 771
Citations - 87172
Anders Björklund is an academic researcher from Lund University. The author has contributed to research in topics: Transplantation & Dopamine. The author has an hindex of 165, co-authored 769 publications receiving 84268 citations. Previous affiliations of Anders Björklund include University of Washington & Institute for the Study of Labor.
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Income inequality and income mobility in the Scandinavian countries compared to the United States
Rolf Aaberge,Anders Björklund,Markus Jäntti,Mårten Palme,Peder J. Pedersen,Nina Smith,Tom Wennemo +6 more
TL;DR: In this paper, the authors compared income inequality and income mobility in the Scandinavian countries and the United States during 1980-90 and found evidence of greater dispersion of first differences of relative earnings and income.
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Intrastriatal glial cell line-derived neurotrophic factor promotes sprouting of spared nigrostriatal dopaminergic afferents and induces recovery of function in a rat model of Parkinson's disease
TL;DR: Results show that intrastriatal glial cell line-derived neurotrophic factor can stimulate substantial axonal sprouting and reinnervation of the partially deafferated striatum to a degree sufficient to reverse the lesion-induced deficit in spontaneous motoric behaviour, indicating that a direct action of glialcell line- derived neurotrophicfactor on spared dopaminergic afferents in the striatum may be important for functional recovery in the rat Parkinson model.
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Connectivity of striatal grafts implanted into the ibotenic acid-lesioned striatum--III. Efferent projecting graft neurons and their relation to host afferents within the grafts.
TL;DR: Efferent projections of intrastriatally implanted striatal neurons have been studied using a combination of anterograde and retrograde axonal tracers to identify neurons labelled with both [3H]thymidine and Fluoro-Gold.
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Neural transplantation--an experimental tool with clinical possibilities.
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Overexpression of Glial Cell Line-Derived Neurotrophic Factor Using a Lentiviral Vector Induces Time- and Dose-Dependent Downregulation of Tyrosine Hydroxylase in the Intact Nigrostriatal Dopamine System
TL;DR: Results indicate that downregulation of TH protein reflects a compensatory effect in response to continuous GDNF stimulation of the DA neurons mediated by a combination of overactivity at the DA synapse and a direct GDNF-induced action on TH gene expression to maintain long-term DA neuron function within the normal range.