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Andrew K. Dunn

Researcher at University of Texas at Austin

Publications -  268
Citations -  14875

Andrew K. Dunn is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Speckle pattern & Cerebral blood flow. The author has an hindex of 52, co-authored 248 publications receiving 12946 citations. Previous affiliations of Andrew K. Dunn include University of York & Nottingham Trent University.

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Intrinsic brain activity triggers trigeminal meningeal afferents in a migraine model

TL;DR: This work establishes a link between migraine aura and headache by demonstrating that cortical spreading depression activates trigeminovascular afferents and evokes a series of cortical meningeal and brainstem events consistent with the development of headache.
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Laser speckle contrast imaging in biomedical optics

TL;DR: The underlying physics of speckle contrast imaging is reviewed, recent developments to improve the quantitative accuracy of blood flow measures are discussed and applications in neuroscience, dermatology and ophthalmology are reviewed.
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Dynamic Imaging of Cerebral Blood Flow Using Laser Speckle

TL;DR: Using this method, dynamic images of the relative CBF changes during focal cerebral ischemia and cortical spreading depression were obtained along with electrophysiologic recordings and validated through direct comparison with conventional laser-Doppler measurements.
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Three dimensional Monte Carlo code for photon migration through complex heterogeneous media including the adult human head

TL;DR: A novel Monte Carlo code for photon migration through 3D media with spatially varying optical properties, known as 'tMCimg', is described and can serve as a resource for solving the forward problem for complex 3D structural data obtained by MRI or CT.
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Ultraflexible nanoelectronic probes form reliable, glial scar–free neural integration

TL;DR: In vivo two-photon imaging and postmortem histological analysis revealed seamless, subcellular integration of NET probes with the local cellular and vasculature networks, featuring fully recovered capillaries with an intact blood-brain barrier and complete absence of chronic neuronal degradation and glial scar.