Anthony Seaton

TL;DR: Exposing the mesothelial lining of the body cavity of mice to long multiwalled carbon nanotubes results in asbestos-like, length-dependent, pathogenic behaviour, including inflammation and the formation of lesions known as granulomas.

TL;DR: It is suggested that ultra-fine particles in the nature of the urban particulate cloud are able to provoke alveolar inflammation, with release of mediators capable, in susceptible individuals, of causing exacerbations of lung disease and of increasing blood coagulability, thus also explaining the observed increases in cardiovascular deaths associated with urban pollution episodes.

TL;DR: The pursuit of responsible nanotechnologies can be tackled through a series of grand challenges, argue Andrew D. Maynard and his co-authors, and if they and their colleagues can rise to these challenges the true extent of any risks involved should become clear.

TL;DR: Available data do not support the concept that low-level exposure to asbestos is a health hazard in buildings and schools, and the concentration of asbestos fibers in air, type of asbestos, and size of fibers must be considered in evaluation of potential health risks.

TL;DR: This present review considers the likely interactions between the ultrafine particles the acute phase response and cardiovascular disease in populations exposed to air pollution episodes.