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Arianne L. Theiss

Researcher at University of Colorado Denver

Publications -  57
Citations -  7491

Arianne L. Theiss is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Prohibitin & Inflammation. The author has an hindex of 23, co-authored 46 publications receiving 6592 citations. Previous affiliations of Arianne L. Theiss include University of North Carolina at Chapel Hill & Scott & White Hospital.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Invasive Escherichia coli are a feature of Crohn's disease

Maiko Sasaki, +10 more
- 30 Jul 2007 - 
TL;DR: IBD-associated E. coli strains from IBD patients regulate cytokine expression and epithelial barrier function, two pathological features of IBD.
Journal ArticleDOI

Tumor Necrosis Factor (TNF) α Increases Collagen Accumulation and Proliferation in Intestinal Myofibroblasts via TNF Receptor 2

Arianne L. Theiss, +3 more
- 28 Oct 2005 - 
TL;DR: It is concluded that TNFα and IGF-I may additively contribute to fibrosis during intestinal inflammation.
Journal ArticleDOI

The role and therapeutic potential of prohibitin in disease.

Arianne L. Theiss, +1 more
- 01 Jun 2011 - 
TL;DR: It is suggested that targeting PHB1 may be a potential therapeutic option for treatment of diseases including cancer, inflammatory bowel disease, insulin resistance/type 2 diabetes, and obesity that are associated with increased oxidative stress and mitochondrial dysfunction.
Journal ArticleDOI

Prohibitin protects against oxidative stress in intestinal epithelial cells

Arianne L. Theiss, +6 more
- 01 Jan 2007 - 
TL;DR: The results suggest that PHB constitutes a previously unrecognized cellular defense against oxidant injury and strategies to modulate PHB levels may constitute a novel therapeutic approach for intestinal inflammatory diseases, wherein oxidative stress plays a critical role in tissue injury and inflammation.