scispace - formally typeset
A

Arindam Bhattacharyya

Researcher at University of Calcutta

Publications -  85
Citations -  1924

Arindam Bhattacharyya is an academic researcher from University of Calcutta. The author has contributed to research in topics: Immune system & Apoptosis. The author has an hindex of 21, co-authored 79 publications receiving 1345 citations. Previous affiliations of Arindam Bhattacharyya include Kalyani Government Engineering College.

Papers
More filters
Journal ArticleDOI

Protease inhibitors from marine actinobacteria as a potential source for antimalarial compound.

TL;DR: The results obtained infer that the peptide from Streptomyces sp LK3 extract possesses anti- Pf activity activity, suggesting that the extracts have novel metabolites and could be considered as a potential source for drug development.
Journal ArticleDOI

Exosomes Produced by Mesenchymal Stem Cells Drive Differentiation of Myeloid Cells into Immunosuppressive M2-Polarized Macrophages in Breast Cancer.

TL;DR: It is demonstrated that exosomes secreted by human and mouse tumor-educated mesenchymal stem cells drive accelerated breast cancer progression by inducing differentiation of monocytic myeloid-derived suppressor cells into highly immunosuppressive M2-polarized macrophages at tumor beds.
Journal ArticleDOI

CXCL13–CXCR5 co-expression regulates epithelial to mesenchymal transition of breast cancer cells during lymph node metastasis

TL;DR: The EMT-inducing potential of CXCL13 is shown as well as the prognostic value of C XCL13–CXCR5 co-expression in primary BC is demonstrated, suggesting that CxCL13-CX CR5–RANKL–Src axis may present a therapeutic target in LNM positive BC patients.
Journal ArticleDOI

Cadmium induces lung inflammation independent of lung cell proliferation: a molecular approach.

TL;DR: The results prove that cadmium causes both inflammation and cell proliferation when applied in a low dose but proliferative changes occur independent of inflammation.
Journal ArticleDOI

An Organoruthenium Anticancer Agent Shows Unexpected Target Selectivity For Plectin.

TL;DR: Plectin targeting shows potential as a strategy to inhibit tumor invasiveness as shown in cultured tumor spheroids while oral administration of plecstatin-1 to mice reduces tumor growth more efficiently in the invasive B16 melanoma than in the CT26 colon tumor model.