A
Armin Kurtz
Researcher at University of Regensburg
Publications - 342
Citations - 14144
Armin Kurtz is an academic researcher from University of Regensburg. The author has contributed to research in topics: Renin–angiotensin system & Kidney. The author has an hindex of 66, co-authored 339 publications receiving 13521 citations. Previous affiliations of Armin Kurtz include Collège de France & Hochschule Hannover.
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Journal ArticleDOI
Macrophages regulate salt-dependent volume and blood pressure by a vascular endothelial growth factor-C–dependent buffering mechanism
Agnes Machnik,Wolfgang Neuhofer,Jonathan Jantsch,Anke Dahlmann,Tuomas Tammela,Katharina Machura,Joon-Keun Park,F. X. Beck,Dominik N. Müller,Wolfgang Derer,Jennifer Goss,Agata Ziomber,Peter Dietsch,Hubertus Wagner,Nico van Rooijen,Armin Kurtz,Karl F. Hilgers,Kari Alitalo,Kai-Uwe Eckardt,Friedrich C. Luft,Dontscho Kerjaschki,Jens Titze +21 more
TL;DR: It is shown that a high-salt diet in rats leads to interstitial hypertonic Na+ accumulation in skin, resulting in increased density and hyperplasia of the lymphcapillary network and VEGFC is identified as an osmosensitive, hypertonicity-driven gene intimately involved in salt-induced hypertension.
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Rate of erythropoietin formation in humans in response to acute hypobaric hypoxia
TL;DR: EPO levels during hypoxia were significantly elevated after 114 and 84 min (3,000 and 4,000 m) and rising thereafter continuously for the period investigated, corresponding to 1.8-fold and 3.0-fold increases in the calculated production rate of the hormone.
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Physiology of Kidney Renin
TL;DR: The protease renin is the key enzyme of the renin-angiotensin-aldosterone cascade, which is relevant under both physiological and pathophysiological settings, and a great number of intrarenally generated or systemically acting factors have been identified.
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Atrial natriuretic peptide inhibits renin release from juxtaglomerular cells by a cGMP-mediated process
TL;DR: The results suggest that ANP inhibits renin release from juxtaglomerular cells by a cGMP-dependent process that does not involve changes in intracellular calcium.
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Connexin40 Is Essential for the Pressure Control of Renin Synthesis and Secretion
TL;DR: It is shown that in the absence of connexin40 (Cx40), which form gap junctions between juxtaglomerular and endothelial cells, the negative control of renin secretion and synthesis by angiotensin II and by intravasal pressure is abrogated, while the regulation by salt intake andrenergic stimulation is maintained.