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Atsushi Hori

Researcher at Juntendo University

Publications -  17
Citations -  59

Atsushi Hori is an academic researcher from Juntendo University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 3, co-authored 13 publications receiving 23 citations. Previous affiliations of Atsushi Hori include Shinshu University & RMIT University.

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Inhibition of BCL2A1 by STAT5 inactivation overcomes resistance to targeted therapies of FLT3-ITD/D835 mutant AML

TL;DR: In this article , a cap analysis gene expression (CAGE) technology revealed that the gene expression of BCL2A1 transcription start sites was increased in primary AML cells bearing D835 in the tyrosine kinase domain (FLT3-ITD/D835), which can induce resistance to TKIs.
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Serum sphingomyelin species profile is altered in hematologic malignancies.

TL;DR: The present study revealed that the serum SM species profile in patients with hematologic malignancies differed from that of normal subjects despite total serum SM and PC concentrations and SM/PC ratios being similar between the various cancer groups and the normal group.
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Rapid quantitative analysis of human serum sphingomyelin species using MALDI-TOF mass spectrometry with lipid hydrolase treatment

TL;DR: A rapid, and quantitative method for identifying serum SM species using lipid hydrolase treatment and MALDI-TOF MS is developed, suitable for clinical laboratory studies to examine the associations between SM species and disease states.
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Hemolysis Is Responsible for Elevation of Serum Iron Concentration After Regular Exercises in Judo Athletes

TL;DR: The results of this study indicate that serum iron concentration increases significantly after Judo exercise due to hemolysis.
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Abnormal Cardiac Repolarization After Seizure Episodes in Structural Brain Diseases: Cardiac Manifestation of Electrical Remodeling in the Brain?

TL;DR: In this article, structural brain abnormalities are correlated with abnormal cardiac repolarization in patients with seizure or epilepsy and can be associated with sudden death, which can be a manifestation of electrophysiological remodeling in the brain.