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Attila T. Lorincz
Researcher at Queen Mary University of London
Publications - 234
Citations - 24010
Attila T. Lorincz is an academic researcher from Queen Mary University of London. The author has contributed to research in topics: Cervical intraepithelial neoplasia & Cervical cancer. The author has an hindex of 76, co-authored 233 publications receiving 23065 citations. Previous affiliations of Attila T. Lorincz include University of California, Santa Barbara & Silver Spring Networks.
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Journal ArticleDOI
The causal relation between human papillomavirus and cervical cancer
TL;DR: It is the right time for medical societies and public health regulators to consider the causal role of human papillomavirus infections in cervical cancer and to define its preventive and clinical implications.
Journal ArticleDOI
The Elevated 10-Year Risk of Cervical Precancer and Cancer in Women With Human Papillomavirus (HPV) Type 16 or 18 and the Possible Utility of Type-Specific HPV Testing in Clinical Practice
Michelle J. Khan,Philip E. Castle,Attila T. Lorincz,Sholom Wacholder,Mark S. Sherman,David R. Scott,Brenda R. Rush,Andrew G. Glass,Mark Schiffman +8 more
TL;DR: HPV screening that distinguishes HPV16 and HPV18 from other oncogenic HPV types may identify women at the greatest risk of > or = CIN3 and may permit less aggressive management of other women with onCogenic HPV infections.
Journal ArticleDOI
Human papillomavirus infection of the cervix: relative risk associations of 15 common anogenital types.
Attila T. Lorincz,Richard Reid,A. B. Jenson,Mitchell D. Greenberg,Wayne D. Lancaster,Robert J. Kurman +5 more
TL;DR: During the years 1982-1989, 2627 women were recruited into eight studies analyzing the relationship between human papillomavirus (HPV) infection and cervical neoplasia, and each cervical sample was rescreened for HPV DNA by low-stringency Southern blot hybridization.
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Analysis of the physical state of different human papillomavirus DNAs in intraepithelial and invasive cervical neoplasm.
TL;DR: It is indicated that detectable integration of HPV DNA, regardless of type, occurs infrequently in cervical intraepithelial neoplasia and the absence of HPV 16 DNA integration in some carcinomas implies that integration is not always required for malignant progression.
Journal ArticleDOI
The causal link between human papillomavirus and invasive cervical cancer: A population-based case-control study in colombia and spain
Nubia Muñoz,F. X. Bosch,S de Sanjosé,Luis Alberto Tafur,Isabel Izarzugaza,Miguel Gili,P. Viladiu,Carmen Navarro,C. Martos,Nieves Ascunce,L. C. Gonzalez,John M. Kaldor,E. Guerrero,Attila T. Lorincz,M. Santamaria,P. Alonso De Ruiz,Nubia Aristizabal,Keerti V. Shah +17 more
TL;DR: There is a very strong association between HPV 16, 18, 31, 33 and 35 and invasive cervical cancer and that this association is probably causal.