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Barbara J. Sahakian

Researcher at University of Cambridge

Publications -  651
Citations -  75147

Barbara J. Sahakian is an academic researcher from University of Cambridge. The author has contributed to research in topics: Cognition & Working memory. The author has an hindex of 145, co-authored 612 publications receiving 69190 citations. Previous affiliations of Barbara J. Sahakian include University of Westminster & Shanghai Jiao Tong University.

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Grand challenges in global mental health

TL;DR: A consortium of researchers, advocates and clinicians announces here research priorities for improving the lives of people with mental illness around the world, and calls for urgent action and investment.
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Stop-signal inhibition disrupted by damage to right inferior frontal gyrus in humans.

TL;DR: This work uses a new observer-independent method to relate the degree of damage within a specific prefrontal region to performance on a stop-signal task that is sensitive to the neurodevelopmental aspects of stopping behavior and to attention-deficit/hyperactivity disorder (ADHD) as well as its amelioration by methylphenidate.
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Traumatic brain injury: integrated approaches to improve prevention, clinical care, and research

Andrew I R Maas, +342 more
- 01 Dec 2017 - 
TL;DR: The InTBIR Participants and Investigators have provided informed consent for the study to take place in Poland.
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Planning and spatial working memory following frontal lobe lesions in man.

TL;DR: Patients with unilateral or bilateral frontal lobe excisions were compared with age and IQ matched controls on a computerized battery of tests of spatial working memory and planning, revealing an impairment of higher cognitive functioning following frontal lobe damage.
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Dissociable Deficits in the Decision-Making Cognition of Chronic Amphetamine Abusers, Opiate Abusers, Patients with Focal Damage to Prefrontal Cortex, and Tryptophan-Depleted Normal Volunteers: Evidence for Monoaminergic Mechanisms

TL;DR: It is suggested that chronic amphetamine abusers show similar decision-making deficits to those seen after focal damage to orbitofrontal PFC, which may reflect altered neuromodulation of the orbitof prefrontal PFC and interconnected limbic-striatal systems by both the ascending 5-HT and mesocortical dopamine projections.