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Bilal Piperdi

Researcher at Merck & Co.

Publications -  125
Citations -  12529

Bilal Piperdi is an academic researcher from Merck & Co.. The author has contributed to research in topics: Pembrolizumab & Lung cancer. The author has an hindex of 32, co-authored 122 publications receiving 9727 citations. Previous affiliations of Bilal Piperdi include University of Texas MD Anderson Cancer Center & University of Maryland Medical Center.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Skin Toxicity Evaluation Protocol With Panitumumab (STEPP), a Phase II, Open-Label, Randomized Trial Evaluating the Impact of a Pre-Emptive Skin Treatment Regimen on Skin Toxicities and Quality of Life in Patients With Metastatic Colorectal Cancer

TL;DR: The incidence of specific >or= grade 2 skin toxicities during the 6-week skin treatment period was reduced by more than 50% in the pre-emptive group compared with the reactive group, and the pre.emptive skin treatment regimen was well tolerated.
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Clinical safety and activity of pembrolizumab in patients with malignant pleural mesothelioma (KEYNOTE-028): preliminary results from a non-randomised, open-label, phase 1b trial

TL;DR: Pembrolizumab appears to be well tolerated and might confer anti-tumour activity in patients with PD-L1-positive malignant pleural mesothelioma, and response durability and efficacy in this patient population warrants further investigation.
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Pembrolizumab in Patients With Extensive-Stage Small-Cell Lung Cancer: Results From the Phase Ib KEYNOTE-028 Study

TL;DR: Pembrolizumab demonstrated promising antitumor activity in patients with pretreated, PD-L1-expressing SCLC and was consistent with the known safety profile in other tumor types.