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Bing Wang

Researcher at Peking University

Publications -  21
Citations -  937

Bing Wang is an academic researcher from Peking University. The author has contributed to research in topics: Attention deficit hyperactivity disorder & Haplotype. The author has an hindex of 15, co-authored 21 publications receiving 925 citations.

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Journal Article

[Association of 5-HT(2A) receptor polymorphism and attention deficit hyperactivity disorder in children].

TL;DR: For the ADHD combined subtype, the T 102T genotype is a protective factor and the T102C genotypes is a risk factor and for the girl with ADHD combinedSub type, the allele C102 is a disease-predisposing gene.
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Family-based and case-control association studies of catechol-O-methyltransferase in attention deficit hyperactivity disorder suggest genetic sexual dimorphism.

TL;DR: The results suggest that the COMT gene exerts some influence on the risk for ADHD in the Han Chinese population, given the potential for Type I error, and require replication before drawing definitive conclusions.
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Association between polymorphisms in serotonin transporter gene and attention deficit hyperactivity disorder in Chinese Han subjects.

TL;DR: The current study assessed the association between ADHD and two variants of the serotonin transporter gene: the 44‐bp deletion/insertion polymorphism (5‐HTTLPR) and the 17 bp‐repeat polymorphism in intron 2 (STin2.VNTR).
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Possible association of the alpha-2A adrenergic receptor gene (ADRA2A) with symptoms of attention-deficit/hyperactivity disorder.

TL;DR: The results provide weak evidence for a possible role of ADRA2A in ADHD symptom expression in the Han Chinese population, which differs quite substantially from the Caucasian population in the frequencies of alleles at these polymorphisms.
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Serotonin 5-HT1B receptor gene and attention deficit hyperactivity disorder in Chinese Han subjects.

TL;DR: Investigation of the A‐161T and G861C polymorphisms in the 5‐HT1B receptor gene in ADHD trios from the Chinese Han population found no association with ADHD but did find a tendency for excess transmission of the 861G allele to offspring with inattentive ADHD.