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Biplab Dasgupta

Researcher at Cincinnati Children's Hospital Medical Center

Publications -  52
Citations -  9691

Biplab Dasgupta is an academic researcher from Cincinnati Children's Hospital Medical Center. The author has contributed to research in topics: AMPK & Neurofibromin 1. The author has an hindex of 28, co-authored 46 publications receiving 8705 citations. Previous affiliations of Biplab Dasgupta include Washington University in St. Louis & Bose Institute.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Nampt/PBEF/visfatin regulates insulin secretion in β cells as a systemic NAD biosynthetic enzyme

Javier R. Revollo, +12 more
- 07 Nov 2007 - 
TL;DR: It is shown that eNampt does not exert insulin-mimetic effects in vitro or in vivo but rather exhibits robust NAD biosynthetic activity, suggesting a vital framework for the regulation of glucose homeostasis.
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Resveratrol stimulates AMP kinase activity in neurons.

Biplab Dasgupta, +1 more
- 24 Apr 2007 - 
TL;DR: It is shown that resveratrol activated AMPK in Neuro2a cells and primary neurons in vitro as well as in the brain, suggesting that neuronal activation of AMPK by resver atrol could affect neuronal energy homeostasis and contribute to the neuroprotective effects of resverAtrol.
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Proteomic Analysis Reveals Hyperactivation of the Mammalian Target of Rapamycin Pathway in Neurofibromatosis 1–Associated Human and Mouse Brain Tumors

Biplab Dasgupta, +4 more
- 01 Apr 2005 - 
TL;DR: It is found that the expression of proteins involved in promoting ribosome biogenesis was increased in the absence of neurofibromin and inhibition of mTOR signaling in Nf1-/- astrocytes abrogated their growth advantage in culture, restoring normal proliferative rates.
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The AMPK Inhibitor Compound C Is a Potent AMPK-Independent Antiglioma Agent

Xiaona Liu, +3 more
- 01 Mar 2014 - 
TL;DR: Compound C is an extremely potent antiglioma agent but it is suggested that caution should be taken in interpreting results when this compound is used as an AMPK inhibitor.