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Camilla H. Stokkevåg

Researcher at Haukeland University Hospital

Publications -  45
Citations -  2133

Camilla H. Stokkevåg is an academic researcher from Haukeland University Hospital. The author has contributed to research in topics: Proton therapy & Relative biological effectiveness. The author has an hindex of 14, co-authored 37 publications receiving 1902 citations. Previous affiliations of Camilla H. Stokkevåg include University of Bergen.

Papers
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Elliptic Flow of Charged Particles in Pb-Pb Collisions at root s(NN)=2.76 TeV

K. Aamodt, +1014 more
TL;DR: In this paper, the first measurement of charged particle elliptic flow in Pb-Pb collisions at root s(NN) p = 2.76 TeV with the ALICE detector at the CERN Large Hadron Collider was performed in the central pseudorapidity region.
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Higher harmonic anisotropic flow measurements of charged particles in Pb-Pb collisions at root s(NN)=2.76 TeV

K. Aamodt, +975 more
TL;DR: The first measurement of the triangular v3, quadrangular v4, and pentagonal v5 charged particle flow in Pb-Pb collisions is reported, and a double peaked structure in the two-particle azimuthal correlations is observed, which can be naturally explained from the measured anisotropic flow Fourier coefficients.
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Charged-Particle Multiplicity Density at Midrapidity in Central Pb-Pb Collisions at root s(NN)=2.76 TeV

K. Aamodt, +990 more
TL;DR: The first measurement of the charged particle multiplicity density at midrapidity in Pb-Pb collisions at a center-of-mass energy per nucleon pair root s(NN) = 2.76 TeV is presented in this paper.
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Rapidity and transverse momentum dependence of inclusive J/Ψ production in pp collisions at s=7 TeV

K. Aamodt, +922 more
- 25 Oct 2011 - 
TL;DR: In this paper, the first results obtained detecting the J/psi through the dilepton decay into e(+)e(-) and mu(+)mu(-) pairs in the rapidity ranges vertical bar y vertical bar < 0.9 and 2.5 < y < 4, respectively, and with acceptance down to zero PT.
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Exploration and application of phenomenological RBE models for proton therapy

TL;DR: There were considerable variations between the estimations of RBE and RBE-weighted doses from the different models and these variations were a consequence of fundamental differences in experimental databases, model assumptions and regression techniques.