scispace - formally typeset
C

Carla Castiglia Gonzaga

Researcher at RMIT University

Publications -  167
Citations -  2118

Carla Castiglia Gonzaga is an academic researcher from RMIT University. The author has contributed to research in topics: Bond strength & Medicine. The author has an hindex of 20, co-authored 153 publications receiving 1692 citations. Previous affiliations of Carla Castiglia Gonzaga include Sao Paulo State University & University of São Paulo.

Papers
More filters
Journal ArticleDOI

Mechanical properties of resin cements with different activation modes.

TL;DR: No correlation was found between flexural strength and hardness, indicating that other factors besides the degree of cure (e.g. filler content and monomer type) affect theflexural strength of composites.
Journal ArticleDOI

Slow crack growth and reliability of dental ceramics.

TL;DR: This study provides a detailed microstructural and slow crack growth characterization of widely used dental ceramics to assist the clinician in choosing the best ceramic material for each situation as well as predicting its clinical longevity.
Journal ArticleDOI

Chlorhexidine diminishes the loss of bond strength over time under simulated pulpal pressure and thermo-mechanical stressing

TL;DR: CXH digluconate at 2% was able to diminish loss of microtensile bond strength over time associated to both etch-and-rinse and self-etch adhesives, and lower concentration of CHX (0.2%) was not able to diminished the loss of bond strengthover time when associated to theSelf-etch adhesive CTSB.
Journal ArticleDOI

Degree of Conversion of a Resin Cement Light-Cured Through Ceramic Veneers of Different Thicknesses and Types

TL;DR: The effect of different types and thicknesses of ceramic veneers on the degree of conversion of a light-cured resin-based cement (RelyX Veneer) depends on the thickness and type of ceramics employed when veneer thicker than 1.5 mm are cemented.
Journal ArticleDOI

Platelet-rich plasma (PRP) impairs the craniofacial bone repair associated with its elevated TGF-β levels and modulates the co-expression between collagen III and α-smooth muscle actin.

TL;DR: The higher levels of TGF‐β associated with the both expression of collagen III and α‐SMA on defect treated with PRP suggest that its biomaterial induce an effect that can be considered similarly to a fibroproliferative disorder.