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Carlo M. Croce

Researcher at Ohio State University

Publications -  1156
Citations -  199822

Carlo M. Croce is an academic researcher from Ohio State University. The author has contributed to research in topics: microRNA & Cancer. The author has an hindex of 198, co-authored 1135 publications receiving 189007 citations. Previous affiliations of Carlo M. Croce include University of Nebraska Medical Center & University of California, Los Angeles.

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The BCSC-1 locus at chromosome 11q23-q24 is a candidate tumor suppressor gene

Eric S. Martin, +9 more
- 30 Sep 2003 - 
TL;DR: The data suggest that BCSC-1 may exert a tumor suppressor activity and is a likely target of the LOH observed on 11q23-q24 in cancer.
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Chronic lymphocytic leukemia of Emu-TCL1 transgenic mice undergoes rapid cell turnover that can be offset by extrinsic CD257 to accelerate disease progression.

Thomas Enzler, +10 more
- 12 Nov 2009 - 
TL;DR: Results indicate that the leukemia cells of TCL1-Tg mice undergo high levels of spontaneous apoptosis that is offset by relatively high rates of leukemia cell proliferation, which might allow for acquisition of mutations that contribute to disease evolution.
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MYC-repressed long noncoding RNAs antagonize MYC-induced cell proliferation and cell cycle progression

Taewan Kim, +5 more
- 09 May 2015 - 
TL;DR: By screening cell cycle-related genes regulated by MYC and the MYC-repressed MYCLos, the GADD45A gene is identified as a target gene of the MYc-repression MYCLs such as MYCLo-4 and MYCLi-6, identified by comparing 3 categories of lncRNAs.
Journal Article

Pathogenetic and Clinical Relevance of MicroRNAs in Colorectal Cancer

Nicola Valeri, +2 more
- 01 Jul 2009 - 
TL;DR: This review will focus on common mechanisms involved in miRNA alterations in CRC, their functional implication in CRC development and the potential use of miRNAs as prognostic and predictive surrogate markers for the management of CRC patients.
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Liver xanthine oxidase increase in mice in three patholgoical models. A possible defence mechanism.

Ezio Tubaro, +4 more
- 01 Jul 1980 - 
TL;DR: A long-lasting, but not permanent, increase in XO activity was observed in mice during bacterial or protozoal infection or with Ehrlich ascitic carcinoma, and a nonspecific defence mechanism is indicated, probably involving enhanced oxidative processes.