Carlos Jesus Pereira Haygert

Bio: Carlos Jesus Pereira Haygert is an academic researcher from Universidade Federal de Santa Maria. The author has contributed to research in topics: Physical examination & Osteoporosis. The author has an hindex of 7, co-authored 23 publications receiving 154 citations.

Automatic Identification of Glaucoma Using Deep Learning Methods.

Abstract: This paper proposes an automatic classification method to detect glaucoma in fundus images The method is based on training a neural network using public image databases The network used in this paper is the GoogLeNet, adapted for this proposal The methodology was divided into two stages, namely: (1) detection of the region of interest (ROI); (2) image classification We first used a sliding-window approach combined with the GoogLeNet network This network was trained using manually extracted ROIs and other fundus image structures Afterwards, another GoogLeNet model was trained using the previous resulting images Then those images were used to train another GoogLeNet model to automatically detect glaucoma To prevent overfitting, data augmentation techniques were used on smaller databases The results demonstrated that the network had a good accuracy, even with poor quality images found in some databases or generated by the data augmentation algorithm

Transverse myelitis associated to HCV infection

Abstract: Complications involving the central nervous system in patients suffering from hepatitis C virus (HCV) infection have been rare. Among them, it appears the transverse myelitis, which has already been reported in likely association with HCV. This paper presents the case study of a 65-year-old woman who developed cervical transverse myelitis linked to chronic HCV infection and anti-HCV antibodies in the spinal fluid, being excluded other etiologies for transverse myelitis. Current evidence has reinforced the likely association between chronic HCV infection and transverse myelitis, especially as recurrent manifestations of the disease. These findings reveal the need for more searching to establish the causal relationship between transverse myelitis and hepatitis C.

Osteomielite esternal por Mycobacterium tuberculosis Sternal osteomyelitis caused by infection with Mycobacterium tuberculosis

Abstract: We report the case of a 74-year-old male patient with a one-year history of chest pain in the suprasternal notch associated with erythema, edema and drainage of purulent material from a fistulous lesion. The patient was HIV-negative with no history of TB. A CT scan of the chest showed an osteolytic lesion in the sternum, and a biopsy revealed caseous granuloma, which, in the microbiological evaluation, was negative for fungi and acid-fast bacilli. The diagnosis of sternal osteomyelitis caused by Mycobacterium tuberculosis was confirmed using PCR.

Bilateral Deep Vein Thrombosis Associated with Inferior Vena Cava Agenesis in a Young Patient Manifesting as Low Back Pain.

Abstract: Congenital absence of the inferior vena cava is a rare vascular anomaly, and most cases are asymptomatic. Nevertheless, patients with inferior vena cava malformations may have increased risk of deep venous thrombosis. Particularly, cases of bilateral deep venous thrombosis may arise owing to an insufficient collateral venous drainage from the lower limbs. We hereby describe a case of a previously healthy young male patient presenting with bilateral lower limb deep venous thrombosis as the initial clinical manifestation of congenital inferior vena cava agenesis. We conclude that in young patients presenting with deep venous thrombosis, especially when thrombosis occurs spontaneously, bilaterally, or recurrently, inferior vena cava anomalies should be thoroughly investigated and ruled out as appropriate.

Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism

Abstract: This is the second edition of this multi-author publication produced by the American Society for Bone and Mineral Research. The volume is divided into very short chapters by more than 80 authors and covers most of the disorders ofbone and mineral metabolism. The problems with a multiauthorship volume are the lack of cohesion between chapters and in the level of expertise devoted to each section. This volume steers a very good compromise between these two extremes. Yes, there is overlap, but this is generally supportive rather than counterproductive. So too are their differences in style. There is, however, a great deal of heterogeneity in the level of referencing and this could be improved. Overall, this variation is not intrusive and is a great credit to the Editor and Associate Editors. The book is exactly what it claims to be a primer. It is a very useful first source of reference for students, physicians and scientists who wish to have a short review of any area in bone metabolism. In the case of physicians, it will be useful in all the many specialties that cover some aspects of bone disease. These include rheumatology, but also endocrinology, nephrology, oncology and gynaecology. The book is compiled mainly by American authors and aimed at the American reader. There are therefore some problems for its wider use. For example, the view that corrected serum calcium should be abandoned, since ionised calcium is available and more appropriate, is clearly not translatable. Perhaps the most difficult area is in therapeutics, as in Europe treatment approaches differ considerably from the USA, particularly in osteoporosis. Agents that are widely available in other countries are relatively neglected, and in some cases ignored. Apart from these deficiencies, I can thoroughly recommed it for all with a passing, or even passionate, interest in bone disease.