C
Carroll E. Cross
Researcher at University of California, Davis
Publications - 275
Citations - 21739
Carroll E. Cross is an academic researcher from University of California, Davis. The author has contributed to research in topics: Ascorbic acid & Lipid peroxidation. The author has an hindex of 70, co-authored 273 publications receiving 21037 citations. Previous affiliations of Carroll E. Cross include University of Pittsburgh & University of California.
Papers
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Journal ArticleDOI
Free radicals, antioxidants, and human disease: where are we now?
Journal ArticleDOI
Oxygen Radicals and Human Disease
Carroll E. Cross,Barry Halliwell,Edward T. Borish,William A. Pryor,Bruce N. Ames,Robert L. Saul,Joe M. McCORD,Denham Harman +7 more
TL;DR: Attention is focused on cigarette smoke oxidants, ischemia-reperfusion-induced radical production, carcinogenesis, and aging, which may well provide a firm foundation for therapeutic breakthroughs in oxy-radical research.
Journal ArticleDOI
Formation of nitric oxide-derived inflammatory oxidants by myeloperoxidase in neutrophils
Jason P. Eiserich,Jason P. Eiserich,Milena Hristova,Carroll E. Cross,A. Daniel Jones,Bruce A. Freeman,Barry Halliwell,Barry Halliwell,Albert van der Vliet +8 more
TL;DR: The data reveal that NO2− may regulate inflammatory processes through oxidative mechanisms, perhaps by contributing to the tyrosine nitration and chlorination observed in vivo.
Journal ArticleDOI
Formation of Reactive Nitrogen Species during Peroxidase-catalyzed Oxidation of Nitrite A POTENTIAL ADDITIONAL MECHANISM OF NITRIC OXIDE-DEPENDENT TOXICITY
TL;DR: The results suggest that NO2−, at physiological or pathological levels, is a substrate for the mammalian peroxidases MPO and lactoperoxidase and that formation of NO2· via per oxidase-catalyzed oxidation ofNO2− may provide an additional pathway contributing to cytotoxicity or host defense associated with increased NO· production.
Journal ArticleDOI
Oxygen-derived species: their relation to human disease and environmental stress.
Barry Halliwell,Carroll E. Cross +1 more
TL;DR: Free radicals and other reactive oxygen species (ROS) are constantly formed in the human body, often for useful metabolic purposes, and antioxidant defenses protect against them, but these defenses are not completely adequate, and systems that repair damage by ROS are also necessary.