C
Christian Veltmann
Researcher at Heidelberg University
Publications - 24
Citations - 1936
Christian Veltmann is an academic researcher from Heidelberg University. The author has contributed to research in topics: Sudden death & Brugada syndrome. The author has an hindex of 14, co-authored 24 publications receiving 1767 citations. Previous affiliations of Christian Veltmann include University of Copenhagen & University of Mannheim.
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Journal ArticleDOI
Long-Term Prognosis of Patients Diagnosed With Brugada Syndrome Results From the FINGER Brugada Syndrome Registry
Vincent Probst,Christian Veltmann,Lars Eckardt,Paola G. Meregalli,Fiorenzo Gaita,Hanno L. Tan,Dominique Babuty,Frederic Sacher,Carla Giustetto,Eric Schulze-Bahr,Martin Borggrefe,M. Haissaguerre,Philippe Mabo,H. Le Marec,Christian Wolpert,Arthur A.M. Wilde +15 more
TL;DR: In the largest series of Brugada syndrome patients thus far, event rates in asymptomatic patients were low and gender, familial history of SCD, inducibility of ventricular tachyarrhythmias during electrophysiological study, and the presence of an SCN5A mutation were not predictive of arrhythmic events.
Journal ArticleDOI
Clinical Aspects and Prognosis of Brugada Syndrome in Children
Vincent Probst,Isabelle Denjoy,Paola G. Meregalli,Jean Christophe Amirault,Frederic Sacher,Jacques Mansourati,Dominique Babuty,Elisabeth Villain,Jacques Victor,Jean-Jacques Schott,Jean Marc Lupoglazoff,Philippe Mabo,Christian Veltmann,Laurence Jesel,Philippe Chevalier,S. A. Clur,Michel Haïssaguerre,Christian Wolpert,Hervé Le Marec,Arthur A.M. Wilde +19 more
TL;DR: In the largest population of children affected by Brugada syndrome described to date, fever represented the most important precipitating factor for arrhythmic events, and as in the adult population, the risk of arrhythmmic events was higher in previously symptomatic patients and in those displaying a spontaneous type I ECG.
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Molecular genetic and functional association of Brugada and early repolarization syndromes with S422L missense mutation in KCNJ8
Hector Barajas-Martinez,Dan Hu,Tania Ferrer,Carlos G. Onetti,Yuesheng Wu,Elena Burashnikov,Madalene Boyle,Tyler Surman,Janire Urrutia,Christian Veltmann,Rainer Schimpf,Martin Borggrefe,Christian Wolpert,Bassiema B. Ibrahim,José A. Sánchez-Chapula,Stephen L. Winters,Michel Haïssaguerre,Charles Antzelevitch +17 more
TL;DR: The hypothesis that KCNJ8 is a susceptibility gene for BrS and ERS is supported and the findings suggest that the S422L-induced gain of function in ATP-sensitive potassium channel current is due to reduced sensitivity to intracellular ATP.
Journal ArticleDOI
Cryoablation Versus Radiofrequency Energy for the Ablation of Atrioventricular Nodal Reentrant Tachycardia (the CYRANO Study) Results From a Large Multicenter Prospective Randomized Trial
Isabel Deisenhofer,Bernhard Zrenner,Yuehui Yin,H. F. Pitschner,Malte Kuniss,Georg Großmann,Sascha Stiller,Armin Luik,Christian Veltmann,Julia Frank,Julia Linner,Heidi Estner,Andreas Pflaumer,Jinjin Wu,Christian von Bary,Ekrem Ücer,Tilko Reents,Stylianos Tzeis,Stephanie Fichtner,Susanne Kathan,Martin R. Karch,Clemens Jilek,Sonia Ammar,Christof Kolb,Zeng-Chang Liu,Bernhard Haller,Claus Schmitt,Gabriele Hessling +27 more
TL;DR: In this paper, a prospective randomized study was conducted to test whether cryoablation is as effective as radiofrequency catheter ablation during both short-term and long-term follow-up with a lower risk of permanent AV block.
Journal ArticleDOI
Effects of flecainide on exercise-induced ventricular arrhythmias and recurrences in genotype-negative patients with catecholaminergic polymorphic ventricular tachycardia
Hiroshi Watanabe,Christian van der Werf,Ferran Rosés-Noguer,Arnon Adler,Naokata Sumitomo,Christian Veltmann,Raphael Rosso,Zahurul A. Bhuiyan,Hennie Bikker,Prince J. Kannankeril,Minoru Horie,Tohru Minamino,Sami Viskin,Bjorn C. Knollmann,Jan Till,Arthur A.M. Wilde +15 more
TL;DR: Fleckcainide was effective in patients with genotype-negative CPVT, suggesting that spontaneous Ca(2+) release from ryanodine channels plays a role in arrhythmia susceptibility, similar to that in Patients with genotypes-positive CPVT.