C
Christine Ellis
Researcher at University of East Anglia
Publications - 9
Citations - 3300
Christine Ellis is an academic researcher from University of East Anglia. The author has contributed to research in topics: Jasmonate & Mutant. The author has an hindex of 8, co-authored 8 publications receiving 3135 citations.
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Journal ArticleDOI
The Jasmonate Signal Pathway
TL;DR: Recent advances in understanding of the role of JAs in plant responses to biotic and abiotic stresses are reviewed.
Journal ArticleDOI
The Arabidopsis mutant cev1 links cell wall signaling to jasmonate and ethylene responses.
TL;DR: It is proposed that the cell wall can signal stress responses in plants, and cev1 mutant phenotype could be reproduced by treating wild-type plants with cellulose biosynthesis inhibitors, and the cellulose synthase mutant rsw1 also had constitutive expression of VSP.
Journal ArticleDOI
The Arabidopsis Mutant cev1 Has Constitutively Active Jasmonate and Ethylene Signal Pathways and Enhanced Resistance to Pathogens
Christine Ellis,John Turner +1 more
TL;DR: It is concluded that cev1 stimulates both the JA and the ethylene signal pathways and that CEV1 regulates an early step in an Arabidopsis defense pathway.
Journal ArticleDOI
COI1 links jasmonate signalling and fertility to the SCF ubiquitin-ligase complex in Arabidopsis.
Alessandra Devoto,Manuela Nieto-Rostro,Daoxin Xie,Christine Ellis,Rebecca Harmston,Elaine Patrick,Jackie Davis,Leigh Sherratt,Mark Coleman,John Turner +9 more
TL;DR: COI1 is expected to form a functional E3-type ubiquitin ligase in plants and to regulate expression of jasmonate responsive genes, possibly by targeted ubiquitination of a histone deacetylase.
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Expression profiling reveals COI1 to be a key regulator of genes involved in wound- and methyl jasmonate-induced secondary metabolism, defence, and hormone interactions.
Alessandra Devoto,Christine Ellis,Andreas Magusin,Hur-Song Chang,Charles Chilcott,Tong Zhu,John Turner +6 more
TL;DR: Results indicate that COI1 plays a pivotal role in wound- and JA signalling and is required for expression of approximately 84% of 212 genes induced by JA and for repression of approximately 46% of 83 genes whose expression was suppressed by wounding.