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Claes-Göran Östenson

Researcher at Karolinska Institutet

Publications -  255
Citations -  12149

Claes-Göran Östenson is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Insulin & Diabetes mellitus. The author has an hindex of 53, co-authored 249 publications receiving 10839 citations. Previous affiliations of Claes-Göran Östenson include Karolinska University Hospital.

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Air pollution and lung cancer incidence in 17 European cohorts : Prospective analyses from the European Study of Cohorts for Air Pollution Effects (ESCAPE)

TL;DR: The meta-analyses showed a statistically significant association between risk for lung cancer and PM10 and PM2·5, and no association between lungcancer and nitrogen oxides concentration or traffic intensity on the nearest street.
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Effects of long-term exposure to air pollution on natural-cause mortality : An analysis of 22 European cohorts within the multicentre ESCAPE project

Rob Beelen, +92 more
- 01 Mar 2014 - 
TL;DR: In this article, the authors investigated the association between natural-cause mortality and long-term exposure to several air pollutants, such as PM2.5, nitrogen oxides, and NOx.
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Pancreatic beta cells are important targets for the diabetogenic effects of glucocorticoids.

TL;DR: Results show that glucocorticoids directly inhibit insulin release in vivo and identify the pancreatic beta cell as an important target for the diabetogenic action of glucoc Corticoids.
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Cancer Incidence in Patients With Type 1 Diabetes Mellitus: A Population-Based Cohort Study in Sweden

TL;DR: Type 1 diabetes is associated with a modest excess cancer risk overall and risks of specific cancers that differ from those associated with type 2 diabetes, including liver, pancreatic, kidney, and endometrial cancer.
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Genetic analysis of non-insulin dependent diabetes mellitus in the GK rat.

TL;DR: A comprehensive study of the genetic basis of diabetes in the Goto-Kakizaki rat, the most widely used animal model of non-obese non-insulin dependent diabetes mellitus, concludes that NIDDM is polygenic and fasting hyperglycaemia and postprandial hyper glycaemia clearly have distinct genetic bases.