Claire E. Hutchison

TL;DR: Most of the AHPs are redundant, positive regulators of cytokinin signaling and affect multiple aspects of plant development, including reduced fertility, increased seed size, reduced vascular development, and a shortened primary root.

TL;DR: The evolutionarily ancient two-component system that is used by cytokinin branches to incorporate a unique family of plant-specific transcription factors is revealed.

TL;DR: The results suggest that CTR1 interacts with ETR1 in vivo, and that this association is required to turn off the ethylene-signaling pathway.

TL;DR: The results indicate that phosphorylation of the receiver domain is required for type-A ARR function and suggest that negative regulation of cytokinin signaling by the type-B ARRs most likely involves phosphorylated-dependent interactions, and show that a subset of the type of ARR proteins are stabilized in response to cytokinIn in part via phosphorylations.

TL;DR: It is shown that high concentrations of cytokinin lead to increased defense responses to a virulent oomycete pathogen, through a process that is dependent on salicylic acid accumulation and activation of defense gene expression, and this results support a model in which cytokin in up-regulates plant immunity via an elevation of SA–dependent defense responses and in which SA in turn feedback-inhibits cytokinIn signaling.