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Claude Bouchard

Researcher at Pennington Biomedical Research Center

Publications -  1105
Citations -  121841

Claude Bouchard is an academic researcher from Pennington Biomedical Research Center. The author has contributed to research in topics: Body mass index & Obesity. The author has an hindex of 153, co-authored 1076 publications receiving 115307 citations. Previous affiliations of Claude Bouchard include Texas A&M University & University of Texas at Austin.

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Metabolic heterogeneity underlying postprandial lipemia among men with low fasting high density lipoprotein cholesterol concentrations.

TL;DR: It is suggested that low HDL cholesterol concentration is a heterogeneous metabolic phenotype that it is not associated with postprandial hyperlipidemia unless accompanied by other features of the insulin resistance syndrome.
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Using MZ twins in experimental research to test for the presence of a genotype-environment interaction effect.

TL;DR: This work proposes a design that involves submitting both members of several MZ twin pairs to an ethically acceptable experimental treatment capable of inducing an adaptative response, and finds that this design with MZ twins may be helpful in the study of human variation for multifactorial phenotypes.
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Inhibition of food intake by inhibitors of fatty acid synthase.

TL;DR: According to a recent report from the World Health Organization, it can be estimated that worldwide about 250 million adults — 7 percent of the population — are obese, defined as having a body-mass index of 25 or more.
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Commingling and segregation analysis of blood pressure in a French-Canadian population.

TL;DR: It is possible that there is a real major gene effect on SBP but that the genotype for elevated SBP has not yet expressed itself in the offspring as they have not yet gone through the risk period, and this possibility needs to be evaluated further in additional studies involving older offspring.
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G-308A polymorphism of the tumor necrosis factor alpha gene promoter and salivary cortisol secretion.

TL;DR: A G-->A polymorphism in the 5' untranslated region of the TNF-alpha gene is associated with elevated morning cortisol levels as well as elevated postprandial cortisol secretion, which might be the endocrine mechanism underlying the previously observed associations between the NcoI T NF-alpha polymorphism and obesity aswell as insulin resistance.