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Claude Bouchard

Researcher at Pennington Biomedical Research Center

Publications -  1105
Citations -  121841

Claude Bouchard is an academic researcher from Pennington Biomedical Research Center. The author has contributed to research in topics: Body mass index & Obesity. The author has an hindex of 153, co-authored 1076 publications receiving 115307 citations. Previous affiliations of Claude Bouchard include Texas A&M University & University of Texas at Austin.

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Why do individuals not lose more weight from an exercise intervention at a defined dose? An energy balance analysis

TL;DR: In this paper, the authors systematically reviewed studies that monitored compliance to exercise prescriptions and measured exercise-induced change in body composition, and concluded that the small magnitude of weight loss observed from the majority of evaluated exercise interventions is primarily due to low doses of prescribed exercise energy expenditures compounded by a concomitant increase in caloric intake.
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Gender difference in postprandial lipemia : importance of visceral adipose tissue accumulation.

TL;DR: Results of the present study suggest that the well known gender difference in visceral adipose tissue accumulation is an important contributing factor involved in the exaggerated postprandial triglyceride-rich lipoprotein response noted in men compared with women.
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Stromelysin-1 and Interleukin-6 Gene Promoter Polymorphisms Are Determinants of Asymptomatic Carotid Artery Atherosclerosis

TL;DR: Genetic factors that predispose to reduced matrix remodeling and to increased inflammation combine to increase susceptibility for intima-media thickening in the carotid bifurcation, a predilection site for atherosclerosis.
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The Human Obesity Gene Map: The 2001 Update

TL;DR: A total of 54 new loci have been added to the human obesity gene map in the past 12 months, and the number of genes, markers, and chromosomal regions that have been associated or linked with human obesity phenotypes is now above 250.
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Abdominal Visceral Fat is Associated with a BclI Restriction Fragment Length Polymorphism at the Glucocorticoid Receptor Gene Locus

TL;DR: The consistent association between the GRL polymorphism detected with BclI and AVF area suggests that this gene or a locus in linkage disequilibrium with the BClI restriction site may contribute to the accumulation of AVF.