C
Claudie Cambon
Researcher at French Institute of Health and Medical Research
Publications - 12
Citations - 921
Claudie Cambon is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Monoamine oxidase & Monoamine neurotransmitter. The author has an hindex of 11, co-authored 12 publications receiving 866 citations.
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Journal ArticleDOI
Oxidative Stress by Monoamine Oxidase Mediates Receptor-Independent Cardiomyocyte Apoptosis by Serotonin and Postischemic Myocardial Injury
Pascale Bianchi,Oxana Kunduzova,Emanuela Masini,Claudie Cambon,Daniele Bani,Laura Raimondi,Marie-Hélène Seguelas,Silvia Nistri,Wilson S. Colucci,Nathalie Leducq,Angelo Parini +10 more
TL;DR: The results supply the first direct evidence that oxidative stress induced by MAO is responsible for receptor-independent apoptotic effects of 5-HT in cardiomyocytes and postischemic myocardial injury.
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Age-dependent increase in hydrogen peroxide production by cardiac monoamine oxidase A in rats
Agnès Maurel,Carole Hernandez,Oksana Kunduzova,Guy Bompart,Claudie Cambon,Angelo Parini,Bernard Frances +6 more
TL;DR: The results suggest that MAO-A could be a major source of H( 2)O(2) in the aging heart.
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Regulation of JNK/ERK activation, cell apoptosis, and tissue regeneration by monoamine oxidases after renal ischemia-reperfusion.
Oxana Kunduzova,Pascale Bianchi,Nathalie Pizzinat,Ghislaine Escourrou,Marie-Hélène Seguelas,Angelo Parini,Claudie Cambon +6 more
TL;DR: The crucial role of MAOs in mediating the production of injurious ROS, which contribute to acute apoptotic and necrotic cell death induced by renal ischemia‐reperfusion in vivo, is demonstrated.
Journal ArticleDOI
Reactive oxygen species production by monoamine oxidases in intact cells.
TL;DR: Results show that, in a normal cell environment, MAO can be a source of reactive oxygen species which could have a functional impact on cell functions and is proposed as a rapid and sensitive procedure to characterize the monoamine oxidase isoforms and their regulation in intact cells.
Journal ArticleDOI
Prevention of apoptotic and necrotic cell death, caspase-3 activation, and renal dysfunction by melatonin after ischemia/reperfusion
Oxana Kunduzova,Ghislaine Escourrou,Marie-Hélène Seguelas,Philippe Delagrange,Claudie Cambon,Angelo Parini +5 more
TL;DR: The demonstration that melatonin prevents postreperfusion apoptotic and necrotic cell death and improves renal function suggests thatMelatonin may represent a novel therapeutic approach for prevention of I/R injury.