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Daisy B. Bautista

Researcher at University of Southern California

Publications -  18
Citations -  1406

Daisy B. Bautista is an academic researcher from University of Southern California. The author has contributed to research in topics: Bronchopulmonary dysplasia & Apnea. The author has an hindex of 16, co-authored 18 publications receiving 1365 citations. Previous affiliations of Daisy B. Bautista include Children's Hospital Los Angeles & Los Angeles County Department of Health Services.

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Journal Article

Obstructive sleep apnea in children with Down syndrome.

TL;DR: It is concluded that children with Down syndrome frequently in have OSAS, with OSA, hypoxemia, and hypoventilation, and it is speculated that OSAS may contribute to the unexplained pulmonary hypertension seen in children with down syndrome.
Journal Article

Clinically unsuspected hypoxia during sleep and feeding in infants with bronchopulmonary dysplasia.

TL;DR: It was concluded that clinically unsuspected oxygen desaturation occurs frequently in preterm infants with and without bronchopulmonary dysplasia, and profound hypoxemia may be responsible for sudden unexplained deaths in these infants.
Journal ArticleDOI

Sudden infant death syndrome in infants of substance-abusing mothers.

TL;DR: It is concluded that ISAM have a higher incidence of SIDS than the non-ISAM general population, however, it was not possible to separate maternal substance abuse from other confounding variables that may also have had an impact on SIDS risk in the ISAM group.
Journal Article

Growth failure in infants with bronchopulmonary dysplasia: nutrition and elevated resting metabolic expenditure.

TL;DR: Infants with bronchopulmonary dysplasia and growth failure have increased metabolic demands and decreased prealbumin values suggesting a relative state of protein-calorie malnutrition.
Journal ArticleDOI

Growth failure in bronchopulmonary dysplasia: elevated metabolic rates and pulmonary mechanics.

TL;DR: It is speculated that growth failure in infants with BPD is partially the result of increased metabolic demands from increased work of breathing but that other mechanisms may act to elevate the metabolic expenditure of these infants.