scispace - formally typeset
D

David Baltimore

Researcher at California Institute of Technology

Publications -  882
Citations -  168784

David Baltimore is an academic researcher from California Institute of Technology. The author has contributed to research in topics: RNA & Virus. The author has an hindex of 203, co-authored 876 publications receiving 162955 citations. Previous affiliations of David Baltimore include Thomas Jefferson University & Johns Hopkins University.

Papers
More filters
Journal ArticleDOI

Activation of the Transcriptional Function of the NF-κB Protein c-Rel by O-GlcNAc Glycosylation

Parameswaran Ramakrishnan, +5 more
- 27 Aug 2013 - 
TL;DR: A stimulus-specific role for hyperglycemia-induced O-GlcNAcylation of c-Rel is suggested in promoting T cell–mediated autoimmunity in conditions such as type 1 diabetes by enhancing the production of T helper cell cytokines.
Journal ArticleDOI

Molecular basis of a mouse strain-specific anti-hapten response

Dennis Y. Loh, +4 more
- 01 May 1983 - 
TL;DR: Comparison of the variation of the closely related VH regions between the two mouse strains showed that there are separate types of evolutionary pressures on the framework and complementarity-determining regions.
Journal ArticleDOI

The Pathway to a Universal Influenza Vaccine

Catharine I. Paules, +4 more
- 17 Oct 2017 - 
TL;DR: A workshop was convened in Rockville, Maryland to identify knowledge gaps in influenza research and develop strategies to fill them and to identify candidates for a universal influenza vaccine.
Journal ArticleDOI

Defective interfering particles of poliovirus. II. Nature of the defect.

Charles N. Cole, +1 more
- 25 May 1973 - 
TL;DR: In this article, it was found that DI particles can carry out most of the standard poliovirus functions including inhibition of cellular macromolecular synthesis, production of viral RNA and production of virus-specific protein.
Journal ArticleDOI

Repression of the IgH enhancer in teratocarcinoma cells associated with a novel octamer factor.

Michael J. Lenardo, +5 more
- 27 Jan 1989 - 
TL;DR: It is proposed that NF-A3 represses specific regulatory sequences that contain the octamer motif, which mediates either negative or positive transcriptional effects, depending on the cell type.