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David T. Armstrong
Researcher at University of Adelaide
Publications - 256
Citations - 13776
David T. Armstrong is an academic researcher from University of Adelaide. The author has contributed to research in topics: Luteinizing hormone & Ovulation. The author has an hindex of 65, co-authored 256 publications receiving 13389 citations. Previous affiliations of David T. Armstrong include University of Toronto & University of Western Ontario.
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Oocyte–somatic cell interactions during follicle development in mammals
TL;DR: Elucidating the nature of oocyte-somatic cell interactions at the various stages of follicle development will have important implications for the understanding of factors regulating folliculogenesis, ovulation rate and fecundity.
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Voltage-activated calcium channels that must be phosphorylated to respond to membrane depolarization.
David T. Armstrong,Roger Eckert +1 more
TL;DR: Evidently the dihydropyridine-sensitive class of voltage-activated calcium channels must be phosphorylated in order to open when the membrane is depolarized, which provides a simple framework for understanding the modulation of calcium channel gating by neurotransmitters, calcium ions, and diHydropyridines.
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Estradiol-17beta biosynthesis in cultured granulosa cells from hypophysectomized immature rats; stimulation by follicle-stimulating hormone.
TL;DR: It is concluded that FSH regulates estrogen biosynthesis in granulosa cells of hypophysectomized rats by a specific stimulation of the aromatizing enzyme system.
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Follicle-stimulating hormone stimulates estradiol-17beta synthesis in cultured Sertoli cells.
TL;DR: These studies provide direct demonstration of estradiol-17beta production by Seroli cells from normal animals, and offer evidence that the synthesis of this steroid is regulated at the level of the aromatizing enzyme system by FSH and adenosine 3':5' cyclic monophosphate.
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Blockade of spontaneous and LH-induced ovulation in rats by indomethacin, an inhibitor of prostaglandin biosynthesis
TL;DR: Indomethacin, an inhibitor of prostaglandin synthesis, blocks ovulation in immature rats pre-treated with pregnant mare serum gonadotropin (PMS), when given either at 0800, 1200 or 1600 hours on the second day after PMS treatment (the equivalent of proestrus in normally cycling adult rats).