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Dermot Kerr
Researcher at Intel
Publications - 117
Citations - 774
Dermot Kerr is an academic researcher from Intel. The author has contributed to research in topics: Computer science & Image processing. The author has an hindex of 9, co-authored 93 publications receiving 480 citations. Previous affiliations of Dermot Kerr include John Radcliffe Hospital & Ulster University.
Papers
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Journal ArticleDOI
DENSE-INception U-net for medical image segmentation.
TL;DR: The experiments highlighted that combining the inception module with dense connections in the U-Net architecture is a promising approach for semantic medical image segmentation.
Proceedings ArticleDOI
Steering a predator robot using a mixed frame/event-driven convolutional neural network
Diederik Paul Moeys,Federico Corradi,Emmett Kerr,Philip Vance,Gautham P. Das,Daniel Neil,Dermot Kerr,Tobi Delbruck +7 more
TL;DR: Although the proposed approach discards the precise DAVIS event timing, it offers the significant advantage of compatibility with conventional deep learning technology without giving up the advantage of data-driven computing.
Posted Content
Steering a Predator Robot using a Mixed Frame/Event-Driven Convolutional Neural Network
Diederik Paul Moeys,Federico Corradi,Emmett Kerr,Philip Vance,Gautham P. Das,Daniel Neil,Dermot Kerr,Tobi Delbruck +7 more
TL;DR: In this paper, a convolutional neural network (CNN) was applied to steer right, left, center and non-visible in a predator/prey scenario using a Dynamic and Active Pixel Sensor (DAVIS) mounted on a robot.
Proceedings ArticleDOI
EAO-SLAM: Monocular Semi-Dense Object SLAM Based on Ensemble Data Association
TL;DR: This work proposes an ensemble data associate strategy for integrating the parametric and nonparametric statistic tests and presents an accurate object pose estimation framework, in which an outliers-robust centroid and scale estimation algorithm and an object pose initialization algorithm are developed to help improve the optimality of pose estimation results.
Journal ArticleDOI
A regulatory circuit that involves HR23B and HDAC6 governs the biological response to HDAC inhibitors.
Maria I. New,Heidi Olzscha,Heidi Olzscha,Heidi Olzscha,Geng Liu,Geng Liu,Geng Liu,Omar Khan,Omar Khan,Omar Khan,Lindsay Stimson,Lindsay Stimson,Lindsay Stimson,Joanna F. McGouran,Dermot Kerr,Amanda S. Coutts,Amanda S. Coutts,Amanda S. Coutts,Benedikt M. Kessler,Mark R. Middleton,N B La Thangue,N B La Thangue,N B La Thangue +22 more
TL;DR: It is shown that HR23B has impacts on two documented effects of HDAC inhibitors;HDAC inhibitors cause apoptosis in cells expressing high levels ofHR23B, whereas in cells with low level expression, HDAC inhibitor treatment is frequently associated with autophagy.