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Devendra K. Agrawal

Researcher at Western University of Health Sciences

Publications -  485
Citations -  15823

Devendra K. Agrawal is an academic researcher from Western University of Health Sciences. The author has contributed to research in topics: Medicine & Inflammation. The author has an hindex of 52, co-authored 411 publications receiving 13447 citations. Previous affiliations of Devendra K. Agrawal include College of Osteopathic Medicine of the Pacific & Creighton University Medical Center.

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Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
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Morphological and biochemical characterization and analysis of apoptosis

TL;DR: The morphology and biochemical features of apoptotic cell death are detailed, with an emphasis on discriminating it from necrosis, and specific and selective techniques which are optimal to target hallmark apoptotic features are described.
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Vitamin D and inflammatory diseases

TL;DR: The potential mechanisms of vitamin D in regulating immune/inflammatory responses in inflammatory diseases are discussed and a pleiotropic role in the pathogenesis of the diseases is considered.
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Mesenchymal stem cells and cutaneous wound healing: novel methods to increase cell delivery and therapeutic efficacy.

TL;DR: The results of the novel studies that show promise for the continued development of MSC-based wound-healing therapies are critically reviewed to provide direction for continued research in this field.
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Insulin‐like growth factor‐1 and TNF‐α regulate autophagy through c‐jun N‐terminal kinase and Akt pathways in human atherosclerotic vascular smooth cells

TL;DR: It is suggested that the expression of autophagy genes can be influenced by IGF‐1 and TNF‐α through c‐jun N‐terminal kinase or Akt pathways and autophileagy might be involved in the regulation of plaque stability.