Author
Diana Brahams
Bio: Diana Brahams is an academic researcher from Lincoln's Inn. The author has contributed to research in topics: Poison control & Injury prevention. The author has an hindex of 17, co-authored 301 publications receiving 2368 citations.
Papers published on a yearly basis
Papers
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TL;DR: Brahms argues that a physician's responsibility to these young patients is greater than that to older girls and that the final decision on whether or not to notify parents should rest with the doctor rather than the patient.
442 citations
170 citations
Journal Article•
TL;DR: Brahams notes that although the judge's remarks should assist physicians treating mentally incapacitated adults, the law in this area is unsatisfactory, and this deficiency in the law might be remedied by restoring parens patriae jurisdiction to the Lord Chancellor.
134 citations
TL;DR: In this article, no cause for action was found for infant McKay's wrongful birth claim against her mother's physician and a health facility whose alleged negligence in detecting Mrs. McKay's rubella and advising an abortion resulted in the plaintiff's being born handicapped.
133 citations
Journal Article•
TL;DR: In Thake v Maurice, a British court ruled that a surgeon was liable for failing to warn a couple of the possibility of returned fertility after a vasectomy, it was held that the birth of a healthy baby was not always a blessing and damages could be awarded for an unwanted birth.
100 citations
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TL;DR: This book is for non-commercial use, as long as it is distributed as a whole in its original form, and the names of the authors and the University of Amsterdam are mentioned.
2,365 citations
TL;DR: This poster presents a poster presented at the 2015 American College of Critical Care Medicine (ACCM) and was endorsed by the Board of Regents of ACCM and the Council of SCCM, and the ASHP Board of Directors.
Abstract: Judith Jacobi, PharmD, FCCM, BCPS; Gilles L. Fraser, PharmD, FCCM; Douglas B. Coursin, MD; Richard R. Riker, MD; Dorrie Fontaine, RN, DNSc, FAAN; Eric T. Wittbrodt, PharmD; Donald B. Chalfin, MD, MS, FCCM; Michael F. Masica, MD, MPH; H. Scott Bjerke, MD; William M. Coplin, MD; David W. Crippen, MD, FCCM; Barry D. Fuchs, MD; Ruth M. Kelleher, RN; Paul E. Marik, MDBCh, FCCM; Stanley A. Nasraway, Jr, MD, FCCM; Michael J. Murray, MD, PhD, FCCM; William T. Peruzzi, MD, FCCM; Philip D. Lumb, MB, BS, FCCM. Developed through the Task Force of the American College of Critical Care Medicine (ACCM) of the Society of Critical Care Medicine (SCCM), in collaboration with the American Society of Health-System Pharmacists (ASHP), and in alliance with the American College of Chest Physicians; and approved by the Board of Regents of ACCM and the Council of SCCM and the ASHP Board of Directors
1,848 citations
TL;DR: Overall, current data provide strong concordant evidence that n-3 PUFA are bioactive compounds that reduce risk of cardiac death.
1,501 citations
TL;DR: According to a stringent random effects model, adherence is most strongly related to outcomes in studies of nonmedication regimens, where measures of adherence are continuous, and where the disease is chronic (particularly hypertension, hypercholesterolemia, intestinal disease, and sleep apnea).
Abstract: Background. Adherence is a factor in the outcome of medical treatment, but the strength and moderators of the adherence-outcome association have not been systematically assessed.Objectives. A quantitative review using meta-analysis of three decades of empirical research correlating adherence with ob
1,417 citations
TL;DR: Within the context of the permissive effect of immunosuppression, genetic changes in HIV-1 may underlie the neuropathological heterogeneity of the AIDS dementia complex and its relatively independent course in relation to the systemic manifestations of AIDS noted in some patients.
Abstract: Infection with human immunodeficiency virus type 1 (HIV-1) is frequently complicated in its late stages by the AIDS dementia complex, a neurological syndrome characterized by abnormalities in cognition, motor performance, and behavior. This dementia is due partially or wholly to a direct effect of the virus on the brain rather than to opportunistic infection, but its pathogenesis is not well understood. Productive HIV-1 brain infection is detected only in a subset of patients and is confined largely or exclusively to macrophages, microglia, and derivative multinucleated cells that are formed by virus-induced cell fusion. Absence of cytolytic infection of neurons, oligodentrocytes, and astrocytes has focused attention on the possible role of indirect mechanisms of brain dysfunction related to either virus or cell-coded toxins. Delayed development of the AIDS dementia complex, despite both early exposure of the nervous system to HIV-1 and chronic leptomeningeal infection, indicates that although this virus is "neurotropic," it is relatively nonpathogenic for the brain in the absence of immunosuppression. Within the context of the permissive effect of immunosuppression, genetic changes in HIV-1 may underlie the neuropathological heterogeneity of the AIDS dementia complex and its relatively independent course in relation to the systemic manifestations of AIDS noted in some patients.
1,245 citations