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Dimitra Kotsopoulos

Researcher at Monash University

Publications -  19
Citations -  1801

Dimitra Kotsopoulos is an academic researcher from Monash University. The author has contributed to research in topics: Blood pressure & Pulse wave velocity. The author has an hindex of 17, co-authored 19 publications receiving 1750 citations. Previous affiliations of Dimitra Kotsopoulos include Monash University, Clayton campus & Monash Medical Centre.

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Dietary soy has both beneficial and potentially adverse cardiovascular effects: a placebo-controlled study in men and postmenopausal women.

TL;DR: In normotensive men and postmenopausal women, soy improved BP and lipids but, overall, did not improve vascular function, and the arterial functional model demonstrated no difference between groups; although again, overall function improved in both groups.
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Non-invasive measurements of arterial structure and function: repeatability, interrelationships and trial sample size.

TL;DR: Under controlled experimental conditions there was good repeatability of measurements of indices between sessions of both intrinsic and functional arterial mechanical properties (central and carotid arterial compliance, intima-media thickness and brachial flow-mediated dilation).
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Evidence that parenteral testosterone therapy may improve endothelium-dependent and-independent vasodilation in postmenopausal women already receiving estrogen

TL;DR: The preliminary data indicate that parenteral testosterone therapy improves both endothelial-dependent (flow-mediated) and endothelium-independent (GTN) brachial artery vasodilation in postmenopausal women using long-term estrogen therapy.
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Effects of Blood Pressure, Smoking, and Their Interaction on Carotid Artery Structure and Function

TL;DR: In conclusion, carotid artery wall remodeling appears to follow Laplace’s law but is insufficient to prevent an increase in circumferential stress in hypertensive subjects.
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Postmenopausal Hormone Replacement Therapy Increases Coagulation Activity and Fibrinolysis

TL;DR: Coagulation activation may partly explain the increases in venous thrombosis and cardiovascular events reported with the use of combined HRT, and there were no associations between changes in hemostatic markers and lipids after treatment.