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Ding-Feng Su
Researcher at Second Military Medical University
Publications - 192
Citations - 6891
Ding-Feng Su is an academic researcher from Second Military Medical University. The author has contributed to research in topics: Blood pressure & Baroreflex. The author has an hindex of 39, co-authored 192 publications receiving 5895 citations. Previous affiliations of Ding-Feng Su include Shenyang Pharmaceutical University.
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NLRP3 inflammasome and its inhibitors: a review.
TL;DR: The present review will describe the structure and mechanisms of activation of the NLRP3 inflammasome, its association with various auto-immune and auto-inflammatory diseases, and the state of research into NLRP2 inflammaome inhibitors.
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Perivascular adipose tissue-derived visfatin is a vascular smooth muscle cell growth factor: role of nicotinamide mononucleotide
TL;DR: A molecular link of visfatin to the paracrine action of PVAT is provided, a novel function of visFatin in promoting VSMC proliferation is demonstrated, and NMN is revealed as a novel signalling molecule that triggers the proliferative process.
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Induction of autophagy contributes to the neuroprotection of nicotinamide phosphoribosyltransferase in cerebral ischemia
TL;DR: The results demonstrate that Nampt promotes neuronal survival through inducing autophagy via regulating TSC2-mTOR-S6K1 signaling pathway in a SIRT1-dependent manner during cerebral ischemia.
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Nicotinamide phosphoribosyltransferase protects against ischemic stroke through SIRT1-dependent adenosine monophosphate-activated kinase pathway.
Pei Wang,Tian-Ying Xu,Yun-Feng Guan,Wei-Wei Tian,Benoit Viollet,Benoit Viollet,Yao-Cheng Rui,Qiwei Zhai,Ding-Feng Su,Chao-Yu Miao,Chao-Yu Miao +10 more
TL;DR: The role of Nampt in brain and stroke remains to be investigated and Nicotinamide phosphoribosyltransferase is implicated in cell fate decisions.
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MicroRNA-124 mediates the cholinergic anti-inflammatory action through inhibiting the production of pro-inflammatory cytokines
TL;DR: It is found that miR-124 is upregulated by cholinergic agonists in LPS-exposed cells and mice and is a potential therapeutic target for the treatment of inflammatory diseases.