D
Divya Kandari
Researcher at Jawaharlal Nehru University
Publications - 9
Citations - 82
Divya Kandari is an academic researcher from Jawaharlal Nehru University. The author has contributed to research in topics: Regulon & Bacillus anthracis. The author has an hindex of 3, co-authored 8 publications receiving 38 citations. Previous affiliations of Divya Kandari include All India Institute of Medical Sciences & Laboratory of Molecular Biology.
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Identification, Functional Characterization, and Regulon Prediction of the Zinc Uptake Regulator (zur) of Bacillus anthracis - An Insight Into the Zinc Homeostasis of the Pathogen.
TL;DR: This study functionally characterizes Zur of B. anthracis and elucidates its role in maintaining zinc homeostasis and an increase in the transcript levels of the regulon genes znuA, rpmG, and yciC upon exposure of cells to TPEN connoted their role in combating hypo-zincemic conditions by bringing about zinc uptake and mobilization.
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Basigin Interacts with Plasmodium vivax Tryptophan-rich Antigen PvTRAg38 as a Second Erythrocyte Receptor to Promote Parasite Growth.
TL;DR: PVTRAg38 binds to two erythrocyte receptors basigin and band 3 through P2 and P4 regions, respectively, to facilitate parasite growth, which can be exploited to develop therapeutics against P. vivax malaria.
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Zur: Zinc-Sensing Transcriptional Regulator in a Diverse Set of Bacterial Species
TL;DR: Zur as discussed by the authors is a regulatory transcriptional factor of the FUR superfamily of proteins, abundant among the bacterial species and known for its intracellular Zn sensing ability.
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Biosensors for the detection of Mycobacterium tuberculosis: a comprehensive overview
TL;DR: This review highlights the recent challenges and scope of improvement in numerous biosensors developed for rapid, specific, selective, and sensitive detection of tuberculosis to reduce the TB burden and successful treatment.
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Insights into the molecular determinants involved in Mycobacterium tuberculosis persistence and their therapeutic implications.
TL;DR: The establishment of persistent infections and the reactivation of persistent bacteria to active bacilli are the two hurdles in effective tuberculosis treatment as mentioned in this paper, which is the two main obstacles in effective TB treatment.