D
Donald M. Lamkin
Researcher at Semel Institute for Neuroscience and Human Behavior
Publications - 25
Citations - 4015
Donald M. Lamkin is an academic researcher from Semel Institute for Neuroscience and Human Behavior. The author has contributed to research in topics: Ovarian cancer & Cancer. The author has an hindex of 14, co-authored 23 publications receiving 3390 citations. Previous affiliations of Donald M. Lamkin include University of Southern Indiana & University of Iowa.
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Associations of Depression With C-Reactive Protein, IL-1, and IL-6 : A Meta-Analysis
TL;DR: Continuity in clinic- and community-based samples suggests there is a dose-response relationship between depression and these inflammatory markers, lending strength to the contention that the cardiac risk conferred by depression is not exclusive to patient populations.
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Paraneoplastic Thrombocytosis in Ovarian Cancer
Rebecca L. Stone,Alpa M. Nick,Iain A. McNeish,Frances R. Balkwill,Hee Dong Han,Justin Bottsford-Miller,Rajesha Rupaimoole,Guillermo N. Armaiz-Pena,Chad V. Pecot,Jermaine Coward,Michael T. Deavers,Hernan G. Vasquez,Diana L. Urbauer,Charles N. Landen,Wei Hu,Hannah Gershenson,Koji Matsuo,Mian M.K. Shahzad,Erin R. King,Ibrahim Tekedereli,Bulent Ozpolat,Edward H. Ahn,Virginia K. Bond,Rui Wang,Angela F. Drew,Francisca C. Gushiken,Donald M. Lamkin,Katherine Collins,Koen DeGeest,Susan K. Lutgendorf,Wah Chiu,Gabriel Lopez-Berestein,Vahid Afshar-Kharghan,Anil K. Sood +33 more
TL;DR: Findings support the existence of a paracrine circuit wherein increased production of thrombopoietic cytokines in tumor and host tissue leads to paraneoplasticThrombocytosis, which fuels tumor growth.
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Inflammation and cancer-related fatigue: Mechanisms, contributing factors, and treatment implications
TL;DR: The current state of the evidence linking inflammation and cancer-related fatigue is examined, drawing from recent human research and from experimental animal models probing effects of cancer and cancer treatment on inflammation and fatigue.
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Biobehavioral Influences on Matrix Metalloproteinase Expression in Ovarian Carcinoma
Susan K. Lutgendorf,Donald M. Lamkin,Nicholas B. Jennings,Jesusa M.G. Arevalo,Frank J. Penedo,Koen DeGeest,Robert R. Langley,Joseph A. Lucci,Steve W. Cole,David M. Lubaroff,Anil K. Sood +10 more
TL;DR: In vitro analyses showed that macrophage MMP-9 production could be directly enhanced by the stress hormones norepinephrine and cortisol by up to a 2-fold increase, which may have implications for patient outcomes in ovarian cancer.
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Chronic stress enhances progression of acute lymphoblastic leukemia via β-adrenergic signaling
Donald M. Lamkin,Erica K. Sloan,Erica K. Sloan,Erica K. Sloan,Ami J Patel,Beverley S Chiang,Matthew A. Pimentel,Jeffrey C.Y. Ma,Jeffrey C.Y. Ma,Jesusa M.G. Arevalo,Jesusa M.G. Arevalo,Kouki Morizono,Steven W. Cole +12 more
TL;DR: Results show that chronic stress can accelerate the progression of human pre-B ALL tumor load via a β-adrenergic signaling pathway that likely involves indirect regulation of ALL biology via alterations in the function of other host cell types such as immune cells or the bone marrow microenvironment.