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E. R. Dickson

Researcher at University of California, Davis

Publications -  10
Citations -  2088

E. R. Dickson is an academic researcher from University of California, Davis. The author has contributed to research in topics: Primary biliary cirrhosis & Pyruvate dehydrogenase complex. The author has an hindex of 7, co-authored 10 publications receiving 1991 citations.

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A model to predict survival in patients with end-stage liver disease

TL;DR: The MELD scale is a reliable measure of mortality risk in patients with end-stage liver disease and suitable for use as a disease severity index to determine organ allocation priorities in patient groups with a broader range of disease severity and etiology.
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Use of a designer triple expression hybrid clone for three different lipoyl domain for the detection of antimitochondrial autoantibodies.

TL;DR: The results indicate that an ELISA using recombinant, cloned autoantigen of pML‐MIT3 is a powerful and very specific method for the detection of AMA.
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Epitope mapping and reactivity of autoantibodies to the E2 component of 2-oxoglutarate dehydrogenase complex in primary biliary cirrhosis using recombinant 2-oxoglutarate dehydrogenase complex

TL;DR: Using overlapping recombinant polypeptides, it was determined that a minimum of 81 amino acids corresponding to the lipoyl domain of OGDC‐E2 are necessary for reactivity, suggesting that a conformational autoepitope is recognized by AMA.
Journal Article

Manifestations of nonsuppurative cholangitis in chronic hepatobiliary diseases

TL;DR: The features of nonsuppurative cholangitis were studied in liver biopsy specimens from 185 patients with chronic active hepatitis (CAH), 280 patients with primary biliary cirrhosis (PBC), and 55 patients with PSC as mentioned in this paper.
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Quantitative Measurement of Autoantibodies to Recombinant Mitochondrial Antigens in Patients With Primary Biliary Cirrhosis: Relationship of Levels of Autoantibodies to Disease Progression

TL;DR: The results show that measurements of AMA by IF or by quantitative EIA methods with recombinant 2‐OADC proteins are not useful parameters for predicting disease progression in patients with PBC.