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Ekaterina M. Panina

Researcher at University of California, Los Angeles

Publications -  10
Citations -  1178

Ekaterina M. Panina is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Regulon & Bacterial genome size. The author has an hindex of 8, co-authored 10 publications receiving 1146 citations. Previous affiliations of Ekaterina M. Panina include Moscow State University.

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BioAmbients: an abstraction for biological compartments

TL;DR: This work presents the BioAmbients calculus, which is suitable for representing various aspects of molecular localization and compartmentalization, including the movement of molecules between compartments, the dynamic rearrangement of cellularcompartments, and the interaction between molecules in a compartmentalized setting.
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Comparative genomics of bacterial zinc regulons: Enhanced ion transport, pathogenesis, and rearrangement of ribosomal proteins

TL;DR: Zinc regulons were predicted to contain a candidate component of the ATP binding cassette, zinT, and candidate AdcR-binding sites were identified upstream of genes encoding pneumococcal histidine triad proteins from a number of pathogenic streptococci, suggesting that PHT proteins are involved in the invasion process.
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Comparative analysis of FUR regulons in gamma-proteobacteria.

TL;DR: The comparative genomics approach is applied to analyze candidate Fur-binding sites in the genomes of Escherichia coli, Salmonella typhi, Yersinia pestis and Vibrio cholerae to predict FUR regulation for several virulence systems.
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A genome-wide screen identifies a Bordetella type III secretion effector and candidate effectors in other species

TL;DR: The screen identified the first TTSS chaperone‐effector locus, btcA‐bteA, and an in vitro interaction between BteA and BtcA was demonstrated, and a large number of novel candidate chaperones and effectors were identified.
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The Bordetella type III secretion system effector BteA contains a conserved N‐terminal motif that guides bacterial virulence factors to lipid rafts

TL;DR: It is hypothesized that BteA associates with the cytoplasmic face of lipid rafts to locally modulate host cell responses to Bordetella attachment, and homologous domains were identified in virulence factors from other bacterial species.