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Elena A. Minina

Researcher at Swedish University of Agricultural Sciences

Publications -  48
Citations -  7845

Elena A. Minina is an academic researcher from Swedish University of Agricultural Sciences. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 19, co-authored 47 publications receiving 6260 citations. Previous affiliations of Elena A. Minina include Russian Academy of Sciences & Heidelberg University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Tudor staphylococcal nuclease is an evolutionarily conserved component of the programmed cell death degradome.

TL;DR: TSN is established as the first biological substrate of metacaspase and demonstrated that despite the divergence of plants and animals from a common ancestor about one billion years ago and their use of distinct PCD pathways, both have retained a common mechanism to compromise cell viability through the cleavage of the same substrate, TSN.
Journal ArticleDOI

Autophagy as initiator or executioner of cell death

TL;DR: A dual role is proposed for autophagy in plant PCD: as an effector of HR PCD lying upstream of the 'point-of-no-return', and also as a downstream mechanism for clearance of terminally differentiated cells during developmental PCD.