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Elisabetta Ferraro

Researcher at University of Turin

Publications -  45
Citations -  7038

Elisabetta Ferraro is an academic researcher from University of Turin. The author has contributed to research in topics: Apoptosome & Programmed cell death. The author has an hindex of 23, co-authored 42 publications receiving 6304 citations. Previous affiliations of Elisabetta Ferraro include University of Eastern Piedmont & University of Rome Tor Vergata.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Chemotherapeutic Drugs and Mitochondrial Dysfunction: Focus on Doxorubicin, Trastuzumab, and Sunitinib

Stefania Gorini, +5 more
- 18 Mar 2018 - 
TL;DR: Therapies targeting and protecting cell metabolism and energy management might be useful tools in protecting muscular tissues against the toxicity induced by chemotherapeutic drugs.
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Autophagic and apoptotic response to stress signals in mammalian cells.

Elisabetta Ferraro, +1 more
- 15 Jun 2007 - 
TL;DR: The interrelations between autophagy and apoptosis under these conditions is discussed, showing that stress is a positive regulator of apoptosis and, in particular, of its apoptosome-mediated mitochondrial pathway.
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Exercise-Induced Skeletal Muscle Remodeling and Metabolic Adaptation: Redox Signaling and Role of Autophagy

Elisabetta Ferraro, +4 more
- 01 Jul 2014 - 
TL;DR: This review has focused its attention on the pathways that are known to mediate physical training-induced plasticity and the involvement of reactive oxygen species, reactive nitrogen species, and autophagy and their controversial effects.
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Endoplasmic Reticulum Stress Induces Apoptosis by an Apoptosome-dependent but Caspase 12-independent Mechanism *

Federica Di Sano, +5 more
- 03 Feb 2006 - 
TL;DR: It is shown here that in the absence of the apoptosome ER stress induces cytochrome c release from the mitochondria but that apoptosis cannot occur, and caspase 12, a protease until now believed to play a central role in the initiation of ER stress-induced cell death in the mouse system, is dispensable for the mitochondrial pathway of death to take place.