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Eun-Kyeong Jo

Researcher at Chungnam National University

Publications -  247
Citations -  20040

Eun-Kyeong Jo is an academic researcher from Chungnam National University. The author has contributed to research in topics: Autophagy & Innate immune system. The author has an hindex of 61, co-authored 228 publications receiving 16506 citations. Previous affiliations of Eun-Kyeong Jo include Konyang University & Imperial College London.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Molecular mechanisms regulating NLRP3 inflammasome activation.

TL;DR: The NLRP3 inflammasome is linked with various human autoinflammatory and autoimmune diseases and may be a promising target for anti-inflammatory therapies, according to current understanding of the mechanisms by which it is activated in the cytosol.
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Vitamin D3 induces autophagy in human monocytes/macrophages via cathelicidin.

TL;DR: It is shown that 1,25-dihydroxyvitamin D3 (1,25D3), the active form of vitamin D, induced autophagy in human monocytes via cathelicidin, which activated transcription of the autophophagy-related genes Beclin-1 and Atg5.
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Upregulated NLRP3 Inflammasome Activation in Patients With Type 2 Diabetes

TL;DR: Data suggest that NLRP3 inflammasome activation is elevated in myeloid cells from type 2 diabetic patients and that antidiabetic treatment with metformin contributes to modulation of inflammaome activation in type 2 diabetes.