Eva Ramos

TL;DR: Melatonin, an ubiquitous and pleiotropic molecule, exerts efficient protection against oxidative stress and ameliorates oxidative/nitrosative damage by a variety of mechanisms, which may contribute in reducing metal‐induced toxicity as postulate here.

TL;DR: It is demonstrated that mycotoxins AFB1, FB1, T2 and OTA disrupt the intestinal barrier permeability by a mechanism involving reduction of claudin isoform expressions, and illite counteracts this disruption.

TL;DR: Experimental evidence supports its actions to ameliorate ischemic long-term behavioural and neuronal deficits, preserving the functional integrity of the blood-brain barrier, inducing neurogenesis and cell proliferation through receptor-dependent mechanism, as well as improving synaptic transmission.

TL;DR: Results suggest that oxidative stress observed is one of the major mechanisms of deltamethrin-induced neurotoxicity and it may be attributed in part to deltAMethrin disposition and metabolism.

TL;DR: The results suggest that oxidative stress observed may be one of the major mechanisms of fipronil-induced neurotoxicity and it may be attributed in part to fiponil disposition and metabolism.