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Fernando J. Peña

Researcher at University of Extremadura

Publications -  169
Citations -  11050

Fernando J. Peña is an academic researcher from University of Extremadura. The author has contributed to research in topics: Sperm & Sperm motility. The author has an hindex of 46, co-authored 154 publications receiving 9837 citations. Previous affiliations of Fernando J. Peña include University of Kentucky & Swedish University of Agricultural Sciences.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Antioxidant supplementation in vitro improves boar sperm motility and mitochondrial membrane potential after cryopreservation of different fractions of the ejaculate.

TL;DR: In the present trial, exogenous Trolox positively affected post-thaw sperm viability (as motility and mitochondrial membrane potential) in both fractions of the ejaculate.
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Assessment of fresh and frozen-thawed boar semen using an Annexin-V assay: a new method of evaluating sperm membrane integrity.

TL;DR: The finding that fewer spermatozoa in Portion I of the ejaculate showed early apoptosis post-freezing, suggests boar spermatozosa in this portion of the seminal plasma are less sensitive to the stress induced by cryopreservation.
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Detection of "apoptosis-like" changes during the cryopreservation process in equine sperm.

TL;DR: It is proposed that sperm mitochondria may be directly involved in the subtle damage that is present in most spermatozoa surviving freezing and thawing.
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Lipid peroxidation, assessed with BODIPY-C11, increases after cryopreservation of stallion spermatozoa, is stallion-dependent and is related to apoptotic-like changes

TL;DR: This LPO is unlikely to represent, per se, a sign of cryopreservation-induced injury, but it is apparently capable of triggering 'apoptotic-like changes' that could result in the sub-lethal cryodamage often seen among surviving spermatozoa.