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Gary A. Dudley

Researcher at University of Georgia

Publications -  146
Citations -  13649

Gary A. Dudley is an academic researcher from University of Georgia. The author has contributed to research in topics: Skeletal muscle & Isometric exercise. The author has an hindex of 61, co-authored 146 publications receiving 13074 citations. Previous affiliations of Gary A. Dudley include Karolinska Institutet & Shepherd Center.

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American College of Sports Medicine Position Stand. Progression Models in Resistance Training for Healthy Adults

TL;DR: In order to stimulate further adaptation toward a specific training goal(s), progression in the type of resistance training protocol used is necessary and emphasis should be placed on multiple-joint exercises, especially those involving the total body.
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Influence of eccentric actions on skeletal muscle adaptations to resistance training.

TL;DR: Three different training regimens were performed to study the influence of eccentric muscle actions on skeletal muscle adaptive responses to heavy resistance exercise, and changes in fibre type composition and capillary frequency were manifest after detraining.
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Influence of complete spinal cord injury on skeletal muscle cross-sectional area within the first 6 months of injury.

TL;DR: There is marked loss of contractile protein early afterSCI which differs among affected skeletal muscles, and it is suggested that the development of muscular imbalance as well as diminution of muscle mass would compromise force potential early after SCI.
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Skeletal muscle atrophy and increased intramuscular fat after incomplete spinal cord injury.

TL;DR: Skeletal muscle atrophy is associated with greater IMF accumulation in SCI group 6 weeks post-injury compared to AB controls, and IMF continues to increase over time in incomplete SCI.
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Influence of complete spinal cord injury on skeletal muscle within 6 mo of injury

TL;DR: The results indicate that vastus lateralis muscle shows marked fiber atrophy, no change in the proportion of type I fibers, and a relative independence of metabolic enzyme levels from activation during the first 24 wk after clinically complete SCI.