scispace - formally typeset
G

George Davey Smith

Researcher at University of Bristol

Publications -  2646
Citations -  294406

George Davey Smith is an academic researcher from University of Bristol. The author has contributed to research in topics: Population & Mendelian randomization. The author has an hindex of 224, co-authored 2540 publications receiving 248373 citations. Previous affiliations of George Davey Smith include Keele University & Western Infirmary.

Papers
More filters
Journal ArticleDOI

Socioeconomic differences in mortality in Britain and the United States.

TL;DR: This editorial considers recent socioeconomic differences in mortality and their causes in the United Kingdom and the United States.
Journal ArticleDOI

Influence of Socioeconomic Circumstances in Early and Later Life on Stroke Risk Among Men in a Scottish Cohort Study

TL;DR: Poorer socioeconomic circumstance was associated with greater stroke risk, with adverse early-life circumstances of particular importance.
Journal ArticleDOI

Cancer and insulin-like growth factor-I: A potential mechanism linking the environment with cancer risk

TL;DR: The associations between insulin-like growth factor-I and prostate cancer, premenopausal breast cancer, and colon cancer are shown, and the effects are sizeable and stronger than the effects seen in relation to most previously reported risk factors.
Journal ArticleDOI

Childhood socioeconomic position and adult cardiovascular mortality : The Boyd Orr Cohort

TL;DR: The relation between childhood socioeconomic position and adult cardiovascular mortality is examined in 3,750 individuals whose families took part in the Carnegie survey of family diet and health in England and Scotland between 1937 and 1939, indicating that the effects of socioeconomic influences upon particular cardiovascular diseases differ according to the age at which they are experienced.
Journal ArticleDOI

Genetic Association of Albuminuria with Cardiometabolic Disease and Blood Pressure

TL;DR: The results suggest therapies that target albuminuria-increasing processes could have antihypertensive effects that are amplified through inhibition of this feed-forward loop, and imply that albuminuria could increase risk of cardiovascular disease through blood pressure.