G
Giuseppe Mancia
Researcher at University of Milano-Bicocca
Publications - 1465
Citations - 152794
Giuseppe Mancia is an academic researcher from University of Milano-Bicocca. The author has contributed to research in topics: Blood pressure & Ambulatory blood pressure. The author has an hindex of 145, co-authored 1369 publications receiving 139692 citations. Previous affiliations of Giuseppe Mancia include University of Milan & Instituto Politécnico Nacional.
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Primary and secondary prevention of stroke by antihypertensive treatment in clinical trials.
TL;DR: Evidence is reviewed that drugs acting on the renin-angiotensin system may exert cerebrovascular protective effects additive to the ones associated with blood pressure reduction on this end point.
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Evidence-based medicine: an educational instrument or a standard for implementation?
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Multiple sampling improves norepinephrine reproducibility in essential hypertension: A comparison with the microneurographic technique
Guido Grassi,Gian Battista Bolla,Gino Seravalle,Fosca Quarti-Trevano,Rita Facchetti,Giuseppe Mancia +5 more
TL;DR: In essential hypertension, the reproducibility of plasma norepinephrine as an adrenergic marker can be substantially improved by performing a nore Pinephrine assay on multiple blood samples.
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Ambulatory blood pressure monitoring and antihypertensive treatment
TL;DR: It will be argued that due to lack of prognostic validity and high cost, ABPM should not be employed routinely in treating hypertensives, but it should always be employed for the evaluation of the efficacy of new hypertensive drugs for which its superiority over sphygmomanometry is indisputable.
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Relationship between sRAGE and eotaxin-3 with CRP in hypertensive patients at high cardiovascular risk.
Colomba Falcone,Maria Paola Buzzi,Sara Bozzini,Chiara Boiocchi,Angela D'Angelo,Sandra Schirinzi,Jasmine Choi,Michael Ochan Kilama,Ciro Esposito,Massimo Torreggiani,Giuseppe Mancia +10 more
TL;DR: This study confirms the robust and widely studied role of CRP as an important marker of vascular inflammation and postulates the possible involvement of sRAGE and eotaxin, 2 novel biomarkers, in CVDs.